Xiaodong Mao
Nanjing Medical University
14 Papers
48 Citations
Xiaodong Mao is an academic researcher from Nanjing Medical University. The author has contributed to research in topics: Islet & Medicine. The author has an hindex of 9, co-authored 11 publications.
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Papers
Anti-inflammatory effect of resveratrol on TNF-alpha-induced MCP-1 expression in adipocytes.
Jian Zhu,Wei Yong,Xiaohong Wu,Ying Yu,Jinghuan Lv,Cui-ping Liu,Xiaodong Mao,Yunxia Zhu,Kuanfeng Xu,Xiao Han,Chao Liu +10 more
TL;DR: Investigation of the effects of resveratrol upon both tumor necrosis factor (TNF)-alpha-induced MCP-1 gene expression and its underlying signaling pathways in 3T3-L1 adipocytes found that it inhibited DNA binding activity of the NF-kappaB complex and subsequently suppressed NF- kappaB transcriptional activity in TNF-alpha-stimulated cells.
161
Reversal of hyperglycemia in diabetic rats by portal vein transplantation of islet-like cells generated from bone marrow mesenchymal stem cells
Xiaohong Wu,Cui-ping Liu,Kuanfeng Xu,Xiaodong Mao,Jian Zhu,Jing-jing Jiang,Dai Cui,Mei Zhang,Yu Xu,Chao Liu +9 more
TL;DR: Rat BM-MSCs could be transdifferentiated into islet-like cells in vitro and portal vein transplantation of islet -like cells could alleviate the hyperglycemia of diabetic rats.
124
Community-based study of the association of subclinical thyroid dysfunction with blood pressure.
Yu Duan,Wen Peng,Xiaodong Wang,Wei Tang,Xiaoyun Liu,Shuhang Xu,Xiaodong Mao,Shang-yong Feng,Yu Feng,You-wen Qin,Kuanfeng Xu,Cui-ping Liu,Chao Liu +12 more
TL;DR: It was concluded that subclinical thyroid dysfunction was not associated with blood pressure and neither subclinical hyperthyroidism nor subclinical hypothyroidism independently predicted increased blood pressure.
52
Postnatal Pancreatic Islet β Cell Function and Insulin Sensitivity at Different Stages of Lifetime in Rats Born with Intrauterine Growth Retardation
TL;DR: It is demonstrated that IUGR rats had both impaired pancreatic development and deteriorated glucose tolerance and insulin sensitivity, which would be the internal causes why they were prone to develop type 2 diabetes.
Chronic activation of neutral ceramidase protects beta-cells against cytokine-induced apoptosis.
TL;DR: The N- CDase pathway is active in INS-1 cells, and the chronic activation of N-CDase is involved in the pathological response of β-cells to cytokines, potentially providing protection against cytokine toxicity.