6 Papers
3 Citations
Xiangqing Xu is an academic researcher from Shandong University of Traditional Chinese Medicine. The author has contributed to research in topics: Medicine & Internal medicine. The author has co-authored 1 publications.
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Papers
The role of T cells in acute ischemic stroke
TL;DR: In this paper , the authors discuss the recent findings regarding the infiltration of T cells into ischemic brain tissue, and the mechanisms governing how T cells can facilitate tissue injury or neuroprotection in AIS.
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The Calcium Channel α2δ1 Subunit: Interactional Targets in Primary Sensory Neurons and Role in Neuropathic Pain.
TL;DR: In this paper, the role of Cavα2δ1 and the associated signaling axis in neuropathic pain conditions is discussed and reviewed and discussed the role and role in peripheral primary neurons.
Electroacupuncture alleviates orofacial allodynia and anxiety-like behaviors by regulating synaptic plasticity of the CA1 hippocampal region in a mouse model of trigeminal neuralgia
Yuqi Jia,Hai-tao Li,Guangzhi Zhang,Hongyun Wu,Si-Shuo Zhang,Hong Zhi,Yahan Wang,Jing Wen Zhu,YiFeng Wang,Xiangqing Xu,Cai-Jun Tian,Wenqiang Cui +11 more
TL;DR: Electroacupuncture modulates synaptic plasticity of hippocampal CA1 neurons, thereby reducing abnormal orofacial pain and anxiety-like behavior and provides evidence for a TN treatment strategy.
A Resting-State Functional Magnetic Resonance Imaging Study of Abnormal Frontoparietal Network Function in Migraine without Aura
Wenqiang Cui,Si-Shuo Zhang,Fei Xu,Hai-tao Li,Hong Zhi,Yahan Wang,Yong-jun Huo,Xiangqing Xu,Hongyun Wu +8 more
TL;DR: In this article , the authors investigated frontoparietal network (FPN) dysfunction in participants with migraine without aura (MwoA) and compared abnormal encephalic regional homogeneity values.
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N-Methyl D-aspartate receptor subtype 2B/Ca2+/calmodulin-dependent protein kinase II signaling in the lateral habenula regulates orofacial allodynia and anxiety-like behaviors in a mouse model of trigeminal neuralgia
TL;DR: Findings suggest that activation of NMDA/CaMKII signaling in the LHb contributes to the occurrence and development of TN and related anxiety-like behaviors, and suppressing the activity of CaMKII+ neurons in the bilateral LHb by targeting NMDA/, may represent a novel strategy for treating pain and anxiety associated with TN.