Xiang Li
Zhengzhou University
9 Papers
3 Citations
Xiang Li is an academic researcher from Zhengzhou University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 2, co-authored 5 publications.
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Papers
Fusobacterium nucleatum reduces METTL3-mediated m6A modification and contributes to colorectal cancer metastasis
Shujie Chen,Lu Zhang,Mengjie Li,Ying Zhang,Meng Sun,Ling-ling Wang,Jiebo Lin,Yun Emma Cui,Qiang Chen,Chenqi Jin,Xiang Li,Boya Wang,Hao Chen,Tianhua Zhou,Liangjing Wang,Chih-Hung Hsu,Wei Zhuo +16 more
TL;DR: In this paper , the authors reveal that Fusobacterium nucleatum (F. nucleatum) induces a dramatic decline of m6A modifications in colorectal cancer cells and patient-derived xenograft (PDX) tissues by downregulation of METTL3, contributing to inducation of CRC aggressiveness.
WTAP-mediated m6A modification modulates bone marrow mesenchymal stem cells differentiation potential and osteoporosis
TL;DR: In this paper , the role of Wilms' tumor 1-associated protein (WTAP), a member of the m6A functional protein family, in regulating BMSCs differentiation remains unknown.
Arginine methylation of PPP1CA by CARM1 regulates glucose metabolism and affects osteogenic differentiation and osteoclastic differentiation
Lu Zhang,Guangjun Jiao,Yu Cui You,Xiang Li,Jincheng Liu,Zhen-Hui Sun,Qinghui Li,Zihan Dai,Jinlong Ma,Hongming Zhou,Gang Li,Chunyang Meng,Yun-Xia Chen +12 more
TL;DR: It is discovered that CARM1 reprogrammed glucose metabolism in osteoblasts and osteoclasts from oxidative phosphorylation to aerobic glycolysis, thereby promoting osteogenic differentiation and inhibiting osteoclastic differentiation.
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WTAP-Mediated m6A RNA Methylation Regulates the Differentiation of Bone Marrow Mesenchymal Stem Cells via the miR-29b-3p/HDAC4 Axis
TL;DR: In this article , the biological role and underlying mechanism of Wilms tumor 1-associated protein (WTAP) in OP and the differentiation of bone marrow mesenchymal stem cells (BMMSCs) were explored.
Bioinformatics Analysis of GFAP as a Potential Key Regulator in Different Immune Phenotypes of Prostate Cancer.
TL;DR: Glial fibrillary acidic protein (GFAP) might serve as a key hub gene within the genetic and epigenetic regulatory networks in prostate cancer and may be a potential biomarker for both PC diagnosis and prognosis.