William E. Munger
Japan Tobacco
20 Papers
313 Citations
William E. Munger is an academic researcher from Japan Tobacco. The author has contributed to research in topics: Hyperplasia & Gene expression. The author has an hindex of 11, co-authored 20 publications. Previous affiliations of William E. Munger include University of North Carolina at Chapel Hill.
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Papers
Molecular Cloning, Genomic Structure, and Expression Analysis of MUC20, a Novel Mucin Protein, Up-regulated in Injured Kidney
Toshio Higuchi,Takuya Orita,Setsuko Nakanishi,Ken Katsuya,Hirotaka Watanabe,Yoshiki Yamasaki,Iwao Waga,Toyomichi Nanayama,Yoshihisa Yamamoto,William E. Munger,Hong-Wei Sun,Ronald J. Falk,J. Charles Jennette,David A. Alcorta,Huiping Li,Tadashi Yamamoto,Yutaka Saito,Motonao Nakamura +17 more
TL;DR: Characteristics suggest that the production of MUC20 is correlated with development and progression of IgAN and other renal injuries, and variations in repeat numbers in the mucin tandem domain, suggest polymorphism of this region.
153
Symptomatic and asymptomatic benign prostatic hyperplasia: Molecular differentiation by using microarrays
Kulkarni Prakash,Gregorio Pirozzi,Michael Elashoff,William E. Munger,Iwao Waga,Rajiv Dhir,Yoshiyuki Kakehi,Robert H. Getzenberg +7 more
TL;DR: To elucidate the molecular differences underlying BPH, gene expression profiles from the prostate transition zone tissue have been analyzed by using microarrays and a set of 511 differentially expressed genes distinguished symptomatic and asymptomatic BPH.
127
Sonic Hedgehog Induces Arteriogenesis in Diabetic Vasa Nervorum and Restores Function in Diabetic Neuropathy
Kengo Fukushima Kusano,Karen L. Allendoerfer,William E. Munger,Roberto Pola,Marta Bosch-Marce,Rudolf Kirchmair,Young Sup Yoon,Cynthia Curry,Marcy Silver,Marianne Kearney,Takayuki Asahara,Douglas W. Losordo +11 more
TL;DR: Data indicate that Shh induces arteriogenesis and restores nerve function in DN, similar to those achieved with vascular endothelial growth factor-2 (VEGF-2) gene therapy.
118
Expression profile of leukocyte genes activated by anti-neutrophil cytoplasmic autoantibodies (ANCA)
Jia Jin Yang,Jia Jin Yang,Gloria A. Preston,Gloria A. Preston,David A. Alcorta,David A. Alcorta,Iwao Waga,Iwao Waga,William E. Munger,William E. Munger,Susan L. Hogan,Susan L. Hogan,Stephen B. Sekura,Stephen B. Sekura,Brian D. Phillips,Brian D. Phillips,Robin P. Thomas,Robin P. Thomas,J. Charles Jennette,J. Charles Jennette,Ronald J. Falk,Ronald J. Falk +21 more
TL;DR: The data indicate that leukocyte genes are activated in vitro by both ANCA Fc and ANCAF(ab')2 pathways and that in vitro activation mimics changes in circulating leukocytes of patients with ANCA disease.
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Microarray studies of gene expression in circulating leukocytes in kidney diseases.
David A. Alcorta,Gloria A. Preston,William E. Munger,Pamela Sullivan,Jia Jin Yang,Iwao Waga,J. Charles Jennette,Ronald Falk +7 more
TL;DR: Genomewide expression profiles of the circulating leukocytes from patients with a variety of renal diseases using the Affymetrix high-density gene chip array technology imply that significant gene expression changes occur in leukocyte that are circulating in patients with renal diseases.
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