Wenxia Ma
University of Alabama at Birmingham
6 Papers
16 Citations
Wenxia Ma is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Downregulation and upregulation & Thermogenesis. The author has an hindex of 3, co-authored 6 publications.
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Papers
MitoQ regulates redox-related noncoding RNAs to preserve mitochondrial network integrity in pressure-overload heart failure.
Seulhee Kim,Jiajia Song,Patrick Ernst,Mary N. Latimer,Chae-Myeong Ha,Kah Yong Goh,Wenxia Ma,Namakkal-Soorappan Rajasekaran,Jianhua Zhang,Xiaoguang Liu,Sumanth D. Prabhu,Gangjian Qin,Adam R. Wende,Martin E. Young,Lufang Zhou +14 more
TL;DR: A unique mechanism by which MitoQ improves myocardial inter-organellar network in HF by maintaining Mfn2 expression via regulation by lncRNAs is supported, which underscores the role of lnc RNAs in the pathogenesis of HF, and the potential of targeting lncRNA as novel therapeutic strategy for HF treatment.
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Ablation of lncRNA Miat attenuates pathological hypertrophy and heart failure.
Liu Yang,Liu Yang,Liu Yang,Jianxin Deng,Wenxia Ma,Aijun Qiao,Shiyue Xu,Yang Yu,Chan Boriboun,Xiang Kang,Dunzheng Han,Patrick Ernst,Lufang Zhou,Jiawei Shi,Eric Zhang,Tao Sheng Li,Hongyu Qiu,Shinichi Nakagawa,Seth Blackshaw,Jianyi Zhang,Gangjian Qin,Gangjian Qin +21 more
TL;DR: In this paper, the conserved long non-coding RNA (lncRNA) myocardial infarction associate transcript (Miat) was identified for its multiple single-nucleotide polymorphisms that are strongly associated with susceptibility to MI, but its role in cardiovascular biology remains elusive.
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Sam68 promotes hepatic gluconeogenesis via CRTC2
Aijun Qiao,Junlan Zhou,Shiyue Xu,Wenxia Ma,Chan Boriboun,Teayoun Kim,Baolong Yan,Jianxin Deng,Liu Yang,Eric Zhang,Yuhua Song,Yongchao C. Ma,Stéphane Richard,Chunxiang Zhang,Hongyu Qiu,Kirk M. Habegger,Jianyi Zhang,Gangjian Qin,Gangjian Qin +18 more
TL;DR: Zhang et al. as discussed by the authors showed that RNA-binding adaptor protein and Src kinase substrate, Sam68, is a novel regulator of hepatic gluconeogenesis and showed that global and hepatic deletions of Sam68 significantly reduce blood glucose levels and the glucagon-induced expression of gluconeogenic genes.
Sam68 impedes the recovery of arterial injury by augmenting inflammatory response.
Shuling Han,Shiyue Xu,Junlan Zhou,Aijun Qiao,Chan Boriboun,Wenxia Ma,Huadong Li,Dauren Biyashev,Liu Yang,Eric Zhang,Qinghua Liu,Shayi Jiang,Ting C. Zhao,Prasanna Krishnamurthy,Chunxiang Zhang,Stéphane Richard,Hongyu Qiu,Jianyi Zhang,Gangjian Qin,Gangjian Qin +19 more
TL;DR: In this article, the role of Src-associated-in-mitosis-68-kDa (Sam68) in cardiovascular biology has not been studied and its functional significance in vascular injury was investigated.
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[Expression of ER, bcl-2, and p53 mRNA in early hybrid embryos of chicken-quail].
He-Rong Liao,Yan Li,Xiao-Ling Guo,Aijun Qiao,Wenxia Ma,Zong-Sheng Zhao,Xiaofeng Zhao,Da-Quan Li,Ningying Xu +8 more
TL;DR: In this paper, the effects of ER, bcl-2, and p53 genes on the early embryonic development for hybrids between chicken and quail were analyzed and the results showed ER mRNA expression of female hybrids were higher than male hybrids from 66 to 84 hatch hours with a highly significant difference.
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