Wenbin Zhu
Fujian Medical University
10 Papers
23 Citations
Wenbin Zhu is an academic researcher from Fujian Medical University. The author has contributed to research in topics: Congenital hypothyroidism & Gestational age. The author has an hindex of 4, co-authored 10 publications.
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Papers
Mutation of glucose-6-phosphate dehydrogenase deficiency in Chinese Han children in eastern Fujian.
Yao Chen,Wenlong Xiu,Yi Dong,Jing Wang,Hong Zhao,Yueqing Su,Jinfu Zhou,Yinglin Zeng,Hua Li,Jingzhi Wo,Feng Lin,Honghua Zhang,Hanqiang Chen,Changyi Yang,Wenbin Zhu +14 more
TL;DR: The proportion ofmutational types in G6PD and the degree of enzyme activity change in various mutational types were found in the neonates of Fujian Province.
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A novel dual oxidase maturation factor 2 gene mutation for congenital hypothyroidism
TL;DR: This study is the first to report a novel c.413-414insA (Y138X) mutation for CH, thereby expanding the mutational spectrum of the DUOXA2 gene.
Perinatal risk factors for congenital hypothyroidism: A retrospective cohort study performed at a tertiary hospital in China.
TL;DR: Perinatal factors should be considered during the diagnosis and treatment of CH, and significant associations were found with regard to parity and the risk of CH in the offspring.
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Risk factors of 125 cases of neonatal congenital hypothyroidism during perinatal period
Jinfu Zhou,Jinying Luo,Hong Zhao,Jing Wang,Feng Lin,Honghua Zhang,Yueqing Su,Yao Chen,Yinglin Zeng,Qingying Lin,Wenbin Zhu +10 more
TL;DR: CH was correlated with mother's age, gestational diabetes, Gestational thyroid disease as well as neonate's birth weight and gestational age, foetus number, fetal distress and other complicated birth defects at certain degree and more attention should be paid to perinatal care.
14
Association between genetic polymorphism of heme oxygenase 1 promoter and neonatal hyperbilirubinemia: a meta-analysis.
TL;DR: A statistically significant association was found between the HMOX1 (GT)n repeat length polymorphism and risk of neonatal hyperbilirubinemia under the allele and under the heterozygous genetic model.
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