Wei Zhang
Sun Yat-sen University
11 Papers
153 Citations
Wei Zhang is an academic researcher from Sun Yat-sen University. The author has contributed to research in topics: Mesenchymal stem cell & Spinal cord injury. The author has an hindex of 9, co-authored 11 publications.
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Papers
Neurotrophin-3 gene modified mesenchymal stem cells promote remyelination and functional recovery in the demyelinated spinal cord of rats
Yu Jiao Zhang,Wei Zhang,Cheng Guang Lin,Ying Ding,Si Fan Huang,Jin Lang Wu,Yan Li,Hongxin Dong,Yuan Shan Zeng +8 more
TL;DR: It is shown that implantation of NT-3 gene-modified MSCs via a recombinant adenoviral vector into a region of ethidium bromide-induced demyelination in the spinal cord resulted in significant improvement of locomotor function and restoration of electrophysiological properties in rats.
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An experimental electro-acupuncture study in treatment of the rat demyelinated spinal cord injury induced by ethidium bromide.
Si-Fan Huang,Ying Ding,Jing-Wen Ruan,Wei Zhang,Jin-Lang Wu,Bing He,Yu-Jiao Zhang,Yan Li,Yuan-Shan Zeng +8 more
TL;DR: Electro-acupuncture treatment can promote NT-3 expression, increase the cell number and differentiation of endogenous OPCs, and remyelination in the demyelinated spinal cord as well as the functional improvement of demyeled spinal cord.
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Electro-acupuncture improves survival and migration of transplanted neural stem cells in injured spinal cord in rats.
TL;DR: The results suggest that the combination of EA and NSCs improves the survival and migration of N SCs in injured spinal cord in rats.
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Morphine acts via μ-opioid receptors to enhance spinal regeneration and synaptic reconstruction of primary afferent fibers injured by sciatic nerve crush
TL;DR: It is demonstrated that morphine may promote the regeneration and synaptic reconstruction of the terminals of injured primary unmyelinated afferent fibers in lamina II of spinal cord, by a process mediated by mu-opioid receptors.
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Inhibition of Mitochondrial Permeability Transition Pore: A Possible Mechanism for Cardioprotection Conferred by Pretreatment with Tanshinone IIA
Shi-zhong Zhang,Zhi-guo Ye,Wei Zhang,Iain C. Bruce,Qiang Xia +4 more
- 01 Jan 2005
TL;DR: It is concluded that pretreatment with tanshinone IIA provides similar cardioprotection to IPC, this effect may be via inhibiting the pore opening during reperfusion, and the mitochondrial ATP sensitive potassium channel may play a role in mediating the por inhibition induced by tansinone IIa.
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