Wei Dai
Wenzhou Medical College
10 Papers
10 Citations
Wei Dai is an academic researcher from Wenzhou Medical College. The author has contributed to research in topics: Medicine & Asthma. The author has an hindex of 3, co-authored 7 publications.
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Papers
Clinical Characteristics of Asymptomatic Patients with SARS-CoV-2 in Zhejiang: An Imperceptible Source of Infection
Wei Dai,Xinmiao Chen,Xiaoting Xu,Zhefeng Leng,Wenwen Yu,Hui Lin,Huiying Li,Jie Lin,Zhangwei Qiu,Yuanrong Dai +9 more
TL;DR: Asymptomatic patients are also infectious; relying only on clinical symptoms, blood cell tests, and radiology examination will lead to misdiagnosis of most patients, leading to the spread of the virus.
Can long-term regular physical activity improve health outcomes among adults with asthma—evidence from a systematic review and Bayesian meta-analysis
TL;DR: PA was effective in improving quality of life (QOL) and pulmonary function and long-term PA appears to be more beneficial for asthmatic patients, and adults with asthma need to carried out regular PA in accordance with the recommendations.
Azithromycin induces apoptosis in airway smooth muscle cells through mitochondrial pathway in a rat asthma model.
TL;DR: In this paper, the effect of AZM on airway smooth muscle cells (ASMCs) proliferation in a rat model of experimental asthma was investigated, and it was shown that AZM increased the apoptosis of ASMCs through a mitochondrial pathway, which might play an important role in ASMs proliferation during asthma remodeling.
CHCHD2 maintains mitochondrial contact site and cristae organizing system stability and protects against mitochondrial dysfunction in an experimental model of Parkinson's disease
Lin Lu,Hengxu Mao,Miaomiao Zhou,Yuwan Lin,Wei Dai,Jiewen Qiu,Yousheng Xiao,Mingshu Mo,Xiaoqin Zhu,Zhuo-Hua Wu,Zhong Pei,Wenyuan Guo,Pingyi Xu,Xiang Chen +13 more
TL;DR: This study found that the overexpression of CHCHD2 can protect against methyl-4-phenylpyridinium (MPP+)-induced mitochondrial dysfunction and inhibit the loss of dopaminergic neurons in the MPTP-induced mouse model.
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Roxithromycin attenuates inflammation via modulation of RAGE-influenced calprotectin expression in a neutrophilic asthma model.
TL;DR: In this paper, the effects of Roxithromycin (RXM) on airway inflammation, the expression of calprotectin, and the activity of the receptor of advanced glycation end products (RAGE) were evaluated.