Walter Zieglgänsberger
Max Planck Society
234 Papers
4.5K Citations
Walter Zieglgänsberger is an academic researcher from Max Planck Society. The author has contributed to research in topics: Excitatory postsynaptic potential & Inhibitory postsynaptic potential. The author has an hindex of 68, co-authored 231 publications. Previous affiliations of Walter Zieglgänsberger include Centre national de la recherche scientifique.
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Papers
Patent
Monitoring neuronal signals
Klaus Dr. Becker,Matthias Eder,Andreas Ranft,Hans-Ulrich Dr. Dodt,Walter Zieglgänsberger +4 more
- 20 Sep 2006
TL;DR: In this article, a method of estimating a parameter representing a quantitative measure of an anaesthesia or coma condition of an individual is described, where the calculating device (20) is adapted for calculating a complexity parameter representing the quantitative measure, and the buffer circuit (21) includes a buffer circuit for storing at least one time series of the neuronal signal and an analysis circuit for subjecting the time series to a recurrence quantification analysis.
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Neuroplasticity in the spinal cord of monoarthritic rats: from metabolic changes to the detection of interleukin-6 using mRNA differential display
Achim Berthele,J. Schadrack,José Manuel Castro-Lopes,Bastian Conrad,Walter Zieglgänsberger,Thomas R. Tölle +5 more
TL;DR: This chapter discusses the neuroplasticity in the spinal cord of monoarthritic rats and the use of the quantitative autoradiographic 2-deoxyglucase technique (2DG) to investigate changes in metabolic activity.
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Neurosteroids do not regulate proopiomelanocortin-gene expression in pituitary cells.
TL;DR: The results suggest that neurosteroids do not control POMC-gene expression at the level of the pituitary corticotroph and exclude significant effects of these compounds at the glucocorticoid receptor.
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Iontophoretically applied morphine and met-enkephalin may interact with different receptors in the brain
Walter Zieglgänsberger,J.P. Fry,Lynn A. Lambert +2 more
- 01 Jan 1980
TL;DR: The finding that the action of met-enkephalin was largely unaltered after the desensitization of the neurone by morphine suggests that different receptors may be involved in their inhibitory actions.
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