Tucker Williamson
Medical University of South Carolina
9 Papers
8 Citations
Tucker Williamson is an academic researcher from Medical University of South Carolina. The author has contributed to research in topics: Zebrafish & Medicine. The author has an hindex of 6, co-authored 8 publications.
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Papers
The cellular and molecular progression of mitochondrial dysfunction induced by 2,4-dinitrophenol in developing zebrafish embryos.
TL;DR: DNP-treated embryos were incapable of sustaining this mitochondrial biogenic response past mid-embryogenesis, as shown by significantly lowered ATP production and ATP levels, decreased gene expression, and the onset of developmental defects.
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Induced Torpor as a Countermeasure for Low Dose Radiation Exposure in a Zebrafish Model.
Thomas J. Cahill,Willian A. da Silveira,Ludivine Renaud,Tucker Williamson,Hao Wang,Dongjun Chung,Ian M. Overton,Sherine S.L. Chan,Gary Hardiman,Gary Hardiman +9 more
TL;DR: In this article, a torpor-like state was induced in zebrafish using melatonin treatment and reduced temperature, and radiation exposure was administered twice over the course of 10 days.
Investigating the effects of chronic low-dose radiation exposure in the liver of a hypothermic zebrafish model
Thomas J. Cahill,Willian A. da Silveira,Ludivine Renaud,Hao Wang,Tucker Williamson,Dongjun Chung,Sherine S.L. Chan,Ian Overton,Gary Hardiman +8 more
TL;DR: In this article , an induced torpor model was developed using the zebrafish to mitigate the deleterious effects of chronic low-dose-rate radiation exposure, and the effects of radiation exposure on this model with a focus on the liver.
Detection of Mitochondrial Toxicity Using Zebrafish
Sherine S.L. Chan,Tucker Williamson +1 more
- 23 Feb 2018
3
Donor Brain Death Exacerbates Complement-Dependent Ischemia/Reperfusion Injury in Transplanted Hearts
Carl Atkinson,Bernhard Floerchinger,Fei Qiao,Sarah Casey,Tucker Williamson,E Moseley,Serban Stoica,Martin Goddard,Xupeng Ge,Stefan G. Tullius,Stephen Tomlinson +10 more
TL;DR: BD exacerbates posttransplantation cardiac ischemia/reperfusion injury in mice and humans and decreases survival of mouse allografts, and targeted complement inhibition in recipients of BD donor hearts ameliorates BD-exacerbated ischemIA/rePerfusions injury.