Tsukasa Koyama
Hokkaido University
294 Papers
3.4K Citations
Tsukasa Koyama is an academic researcher from Hokkaido University. The author has contributed to research in topics: Serotonin & Dopamine. The author has an hindex of 48, co-authored 294 publications.
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Papers
SSRIs and conditioned fear
TL;DR: Behavioral data provide a new explanation of neurochemical adaptations to contextual conditioned fear: increased 5-HT transmission seems to decrease, not increase, fear.
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Generalized spike-wave discharges involve a default mode network in patients with juvenile absence epilepsy : A MEG study
Kotaro Sakurai,Youji Takeda,Naoaki Tanaka,Tsugiko Kurita,Hideaki Shiraishi,Fumiya Takeuchi,Shingo Nakane,Keitaro Sueda,Tsukasa Koyama +8 more
TL;DR: Cortical regions that constitute a default mode network are strongly involved in the GSW process in some patients with JAE, and dSPM shows that focal cortical activation appears at the onset of a GSW.
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Monoamine oxidase inhibitors reduce conditioned fear stress-induced freezing behavior in rats.
Yuji Maki,Takeshi Inoue,Takeshi Izumi,Ihoko Muraki,Koichi Ito,Yuji Kitaichi,Xiaobai Li,Tsukasa Koyama +7 more
TL;DR: Results suggest that acute inhibition of both monoamine oxidase A and B reduced anxiety or fear, while inhibition of monoamine oxidation A or B alone failed to reduce anxiety orFear.
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Context determines the type of sensitized behaviour: a brief review and a hypothesis on the role of environment in behavioural sensitization.
TL;DR: According to the proposed hypothesis, environment does not have a causal role in the development of sensitization, but it modifies the mode of expression of the sensitized behaviour, which results in augmentation of different behaviours.
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Target brain sites of the anxiolytic effect of citalopram, a selective serotonin reuptake inhibitor.
TL;DR: Systemic administration of citalopram attenuated contextual CFS-induced c-Fos expression in the secondary motor cortex, primary somatosensory cortex, and basolateral nucleus of the amygdala is a likely candidate region in which cITALopram exerts its anxiolytic effect.
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