Toshikazu Kondo
Wakayama Medical University
202 Papers
682 Citations
Toshikazu Kondo is an academic researcher from Wakayama Medical University. The author has contributed to research in topics: Medicine & Chemokine. The author has an hindex of 40, co-authored 178 publications. Previous affiliations of Toshikazu Kondo include Ludwig Maximilian University of Munich & Osaka City University.
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Papers
Immune Cells Profiles in the Different Sites of COVID-19-Affected Lung Lobes in a Single Patient
Sadahiro Iwabuchi,Kyohei Miyamoto,Mayuko Hatai,Yurina Mikasa,Masahiro Katsuda,Shin-ichi Murata,Toshikazu Kondo,Hiroki Yamaue,Shinichi Hashimoto +8 more
TL;DR: This analysis of lung samples from a patient helps to determine the COVID-19 pathophysiology at a specific time point, and the virus localization and inflammatory responses at each site of the lungs provide various important indications.
Differential diagnosis between freshwater drowning and saltwater drowning based on intrapulmonary aquaporin-5 expression.
TL;DR: Observations imply that AQP5 expression in type I alveolar epithelial cells was suppressed by hypotonic water to prevent hemodilution from the physiological aspect.
Increased plaque rupture forms peak incidence of acute myocardial infarction in winter.
Yosuke Katayama,Atsushi Tanaka,Akira Taruya,Manabu Kashiwagi,Tsuyoshi Nishiguchi,Yuichi Ozaki,Yasutsugu Shiono,Kunihiro Shimamura,Hironori Kitabata,Takashi Kubo,Takeshi Hozumi,Yuko Ishida,Toshikazu Kondo,Takashi Akasaka +13 more
TL;DR: The peak incidence of AMI in the winter is formed by increased plaque rupture, suggesting environmental temperature has an influence on the pathogenesis of AMi.
Crucial involvement of the CX3CR1-CX3CL1 axis in dextran sulfate sodium-mediated acute colitis in mice.
Feodora I. Kostadinova,Tomohisa Baba,Yuko Ishida,Toshikazu Kondo,Boryana K. Popivanova,Naofumi Mukaida +5 more
TL;DR: Observations would indicate that the CX3CL1‐CX3CR1 axis can regulate the expression of iNOS, a crucial mediator of DSS‐induced colitis, and targeting this axis may be effective for the treatment of IBDs such as UC.
Blockade of IP-10/CXCR3 Promotes Progressive Renal Fibrosis
Izaya Nakaya,Takashi Wada,Kengo Furuichi,Norihiko Sakai,Kiyoki Kitagawa,Hitoshi Yokoyama,Yuko Ishida,Toshikazu Kondo,Takeshi Sugaya,Hiroshi Kawachi,Fujio Shimizu,Shosaku Narumi,Makoto Haino,Craig Gerard,Kouji Matsushima,Shuichi Kaneko +15 more
TL;DR: The results suggest that blockade of IP-10 via CXCR3 contributes to renal fibrosis, possibly by upregulation of transforming growth factor-β1, concomitant with downregulation of hepatocyte growth factor.