Tomoya Kitayama
Mukogawa Women's University
26 Papers
132 Citations
Tomoya Kitayama is an academic researcher from Mukogawa Women's University. The author has contributed to research in topics: Neuropathic pain & Allodynia. The author has an hindex of 11, co-authored 25 publications. Previous affiliations of Tomoya Kitayama include Life Sciences Institute & Hiroshima University.
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Papers
Analgesic action of nicotine on tibial nerve transection (TNT)-induced mechanical allodynia through enhancement of the glycinergic inhibitory system in spinal cord
Md. Joynal Abdin,Norimitsu Morioka,Katsuya Morita,Tomoya Kitayama,Shigeo Kitayama,Toshikatsu Nakashima,Toshihiro Dohi +6 more
TL;DR: Results indicate that the alpha4beta2 and alpha7 nAChR system, by enhancing the activities of glycinergic neurons at the spinal level, exerts a suppressive effect on the nociceptive transduction in neuropathic pain.
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P2X(7) receptor stimulation in primary cultures of rat spinal microglia induces downregulation of the activity for glutamate transport.
Norimitsu Morioka,Md. Joynal Abdin,Tomoya Kitayama,Katsuya Morita,Yoshihiro Nakata,Toshihiro Dohi +5 more
TL;DR: Results suggest that the activation of P2X7 receptors in spinal microglia is important in the regulation of glutamate transport via activation of the extracellular signal‐regulated kinase cascade and production of oxidants.
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The Role of K+-Cl−-Cotransporter-2 in Neuropathic Pain
TL;DR: The hypothesis that an alteration of KCC2 expression has a crucial influence on the initiation/development or maintenance of neuropathic pain is discussed and it is suggested that the alteration of inhibitory signals is dependent on the time after peripheral nerve injury.
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Cyclic ADP-ribose requires FK506-binding protein to regulate intracellular Ca2+ dynamics and catecholamine release in acetylcholine-stimulated bovine adrenal chromaffin cells.
TL;DR: Evidence is presented that FKBP12.6 associated with RyR may be required for Ca2+ release induced by cADPR in bovine adrenal chromaffin cells, which is essential for ACh-stimulated CA release.
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The regulation of glycine transporter GLYT1 is mainly mediated by protein kinase Cα in C6 glioma cells
Norimitsu Morioka,Joynal Md. Abdin,Katsuya Morita,Tomoya Kitayama,Yoshihiro Nakata,Toshihiro Dohi +5 more
TL;DR: In this paper, the mechanism of the phorbol 12-myristate 13-acetate (PMA)-suppressed uptake of glycine in C6 glioma cells which have the native expression of GLYT1 was investigated.
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