Thomas Verrier
French Institute of Health and Medical Research
4 Papers
Thomas Verrier is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Innate lymphoid cell & Chemokine receptor. The author has an hindex of 4, co-authored 4 publications. Previous affiliations of Thomas Verrier include Paris Diderot University & Pasteur Institute.
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Papers
Notch signaling in group 3 innate lymphoid cells modulates their plasticity
Sylvestre Chea,Sylvestre Chea,Sylvestre Chea,Thibaut Perchet,Thibaut Perchet,Thibaut Perchet,Maxime Petit,Maxime Petit,Maxime Petit,Thomas Verrier,Thomas Verrier,Delphine Guy-Grand,Elena Gaia Banchi,Elena Gaia Banchi,Elena Gaia Banchi,Christian A. J. Vosshenrich,Christian A. J. Vosshenrich,James P. Di Santo,James P. Di Santo,Ana Cumano,Ana Cumano,Ana Cumano,Rachel Golub,Rachel Golub,Rachel Golub +24 more
TL;DR: It is shown that the population of natural cytotoxicity receptor–positive ILC3s in mice is composed of two subsets that have distinct developmental requirements, and Notch signaling contributed to the transition of NCR− cells into NCR+ cells, the more proinflammatory subset, in a cell-autonomous manner.
Developmental options and functional plasticity of innate lymphoid cells.
Ai Ing Lim,Ai Ing Lim,Thomas Verrier,Thomas Verrier,Christian A. J. Vosshenrich,Christian A. J. Vosshenrich,James P. Di Santo,James P. Di Santo +7 more
TL;DR: These new results suggest that while ILC differentiation may be 'fixed' in principle, it remains 'flexible' in practice, and a more comprehensive knowledge in the molecular mechanisms that regulate ILC development and effector functions may allow for therapeutic manipulation of ILCs for diverse disease conditions.
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Phenotypic and Functional Plasticity of Murine Intestinal NKp46+ Group 3 Innate Lymphoid Cells
Thomas Verrier,Thomas Verrier,Thomas Verrier,Naoko Satoh-Takayama,Naoko Satoh-Takayama,Nicolas Serafini,Nicolas Serafini,Solenne Marie,Solenne Marie,James P. Di Santo,James P. Di Santo,Christian A. J. Vosshenrich,Christian A. J. Vosshenrich +12 more
TL;DR: An unexpected phenotypic instability within NKp46+ ILC3 is identified that suggests a major role for environmental signals in tuning I LC3 functional plasticity.
The chemokine receptor CXCR6 controls the functional topography of interleukin-22 producing intestinal innate lymphoid cells
Naoko Satoh-Takayama,Naoko Satoh-Takayama,Nicolas Serafini,Nicolas Serafini,Thomas Verrier,Thomas Verrier,Abdessalem Rekiki,Abdessalem Rekiki,Jean-Christophe Renauld,Gad Frankel,James P. Di Santo,James P. Di Santo +11 more
TL;DR: A critical CXCL16-CXCR6 crosstalk is delineated that coordinates the intestinal topography of IL-22 secretion required for mucosal defense and explains the sensitivity of Cxcr6(-/-) mice to C. rodentium.