Thomas P. Batchelor
West Virginia University
19 Papers
27 Citations
Thomas P. Batchelor is an academic researcher from West Virginia University. The author has contributed to research in topics: Medicine & Inhalation. The author has an hindex of 7, co-authored 9 publications.
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Papers
Maternal titanium dioxide nanomaterial inhalation exposure compromises placental hemodynamics.
Alaeddin B. Abukabda,Elizabeth C. Bowdridge,Carroll R. McBride,Thomas P. Batchelor,William T. Goldsmith,Krista L Garner,Sherri Friend,Timothy R. Nurkiewicz,Timothy R. Nurkiewicz +8 more
TL;DR: The results indicate that maternal gestational pulmonary nano‐TiO2 exposure increases placental vascular resistance and impairs umbilical vascular reactivity.
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Group II innate lymphoid cells and microvascular dysfunction from pulmonary titanium dioxide nanoparticle exposure.
Alaeddin B. Abukabda,Carroll R. McBride,Thomas P. Batchelor,William T. Goldsmith,Elizabeth C. Bowdridge,Krista L Garner,Sherri Friend,Timothy R. Nurkiewicz,Timothy R. Nurkiewicz +8 more
TL;DR: These results provide evidence that the uterine microvascular dysfunction that follows pulmonary ENM exposure may be initiated via activation of lung-resident ILC2 and subsequent systemic Th2-dependent inflammation.
Maternal Engineered Nanomaterial Inhalation During Gestation Disrupts Vascular Kisspeptin Reactivity.
Elizabeth C. Bowdridge,Alaeddin B. Abukabda,Kevin J Engles,Carroll R. McBride,Thomas P. Batchelor,William T. Goldsmith,Krista L Garner,Sherri Friend,Timothy R. Nurkiewicz,Timothy R. Nurkiewicz +9 more
TL;DR: There is initial evidence that pulmonary maternal ENM exposure perturbs the normal gestational endocrine vascular axis via a kisspeptin-dependent mechanism, and decreased placental, which may adversely affect health outcomes.
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Interleukin(IL)-1 Regulates Ozone-enhanced Tracheal Smooth Muscle Responsiveness by Increasing Substance P (SP) Production in Intrinsic Airway Neurons of Ferret
TL;DR: Findings show that IL-1 is released during ozone exposure enhances airway responsiveness by modulating SP expression in airway neurons.
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Biological effects of inhaled hydraulic fracturing sand dust. IV. Pulmonary effects
Kristen A. Russ,Janet A. Thompson,Jeffrey S. Reynolds,Robert R. Mercer,Dale W. Porter,Walter McKinney,Richard D. Dey,Mark Barger,Jared L. Cumpston,Thomas P. Batchelor,Michael L. Kashon,Vamsi Kodali,Mark Jackson,Krishnan Sriram,Jeffrey S. Fedan +14 more
TL;DR: Fracking sand dust (FSD) 8 was without large effect on most parameters, and was not pro-inflammatory, as judged histologically and in cultured epithelial cells, but increased reactivity to inhaled MCh at some post-exposure time points and affected Na+ transport in airway epithelium cells.
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