Thomas M. Petro
University of Nebraska Medical Center
59 Papers
512 Citations
Thomas M. Petro is an academic researcher from University of Nebraska Medical Center. The author has contributed to research in topics: Cytokine & Immune system. The author has an hindex of 21, co-authored 55 publications. Previous affiliations of Thomas M. Petro include University of Nebraska Medical Center College of Dentistry & University of Nebraska–Lincoln.
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Papers
Arginine-Induced Germ Tube Formation in Candida albicans Is Essential for Escape from Murine Macrophage Line RAW 264.7
Suman Ghosh,Dhammika H. M. L. P. Navarathna,David D. Roberts,Jake T. Cooper,Audrey L. Atkin,Thomas M. Petro,Kenneth W. Nickerson +6 more
TL;DR: Metabolism of arginine by C. albicans controls hyphal switching and provides an important mechanism for escaping host defense.
Nickel release from orthodontic arch wires and cellular immune response to various nickel concentrations.
Wenyi Jia,Mark W. Beatty,Richard A. Reinhardt,Thomas M. Petro,Donald M. Cohen,Constance R. Maze,E. A. Strom,Melvin Hoffman +7 more
TL;DR: The maximum amount of nickel released from all tested arch wires was 700 times lower than the concentrations necessary to elicit cytotoxic reactions in human PBMCs.
145
Gingival Cell IL‐2 and IL‐4 in Early‐Onset Periodontitis
Scott S. Manhart,Richard A. Reinhardt,Jeffrey B. Payne,Gregory J. Seymour,Erica Gemmell,John K. Dyer,Thomas M. Petro +6 more
TL;DR: The purpose of this study was to compare, using cell blot analysis, the association of gingival tissue mononuclear cells (GTMC) isolated from lesions displaying histories of early-onset periodontitis and gingivitis with the B-cell stimulating cytokine, interleukin (IL)-4, and the T- cell stimulating cytokines, IL-2.
100
Decreased severity of experimental autoimmune encephalomyelitis during resveratrol administration is associated with increased IL-17+IL-10+ T cells, CD4- IFN-γ+ cells, and decreased macrophage IL-6 expression.
Toby J. Imler,Thomas M. Petro +1 more
TL;DR: Protecting T cells which simultaneously produce IL-17 and IL-10 or infiltrating CD4(-) NKT cells that produce IFN-gamma protect against EAE, and adopting transfer of day 14 EAE encephalogenic T cells into mice fed resveratrol reduced the severity of EAE.
98
Exogenous farnesol interferes with the normal progression of cytokine expression during candidiasis in a mouse model.
Dhammika H. M. L. P. Navarathna,Kenneth W. Nickerson,Gerald E. Duhamel,Thomas R. Jerrels,Thomas M. Petro +4 more
TL;DR: The role of farnesol in systemic candidiasis is likely due to its ability to inhibit the critical Th1 cytokines IFN-γ and IL-12 and perhaps to enhance a Th2 cytokine, IL-5.