Teiji Tominaga
Tohoku University
1006 Papers
4.4K Citations
Teiji Tominaga is an academic researcher from Tohoku University. The author has contributed to research in topics: Medicine & Moyamoya disease. The author has an hindex of 55, co-authored 923 publications. Previous affiliations of Teiji Tominaga include Iwate Medical University & Karolinska Institutet.
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Papers
Hyperperfusion in the thalamus on arterial spin labelling indicates non-convulsive status epilepticus
Satoru Ohtomo,Hiroshi Otsubo,Hiroaki Arai,Yoshiteru Shimoda,Yoichiro Homma,Teiji Tominaga +5 more
- 16 Jan 2021
TL;DR: In this paper, arterial spin labeling and EEG were used for the diagnosis of non-convulsive status epilepticus and the association between periodic discharges and rhythmic delta activity in addition to ictal epileptiform discharges was established.
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Subarachnoid small vein occlusion due to inflammatory fibrosis-a possible mechanism for cerebellar infarction in cryptococcal meningoencephalitis: a case report.
TL;DR: C. neoformans can induce extensive fibrosis of the subarachnoid space, which may compress small veins mechanically inducing venule congestion and massive cerebral infarction in such cases, which can be severe and even rapidly fatal.
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Somatic BRAF c.1799T>A p.V600E Mosaicism syndrome characterized by a linear syringocystadenoma papilliferum, anaplastic astrocytoma, and ocular abnormalities.
Yuko Watanabe,Kosuke Shido,Tetsuya Niihori,Hidetaka Niizuma,Yu Katata,Chie Iizuka,Daiju Oba,Kunihiko Moriya,Yuka Saito-Nanjo,Masaei Onuma,Takeshi Rikiishi,Yoji Sasahara,Mika Watanabe,Setsuya Aiba,Ryuta Saito,Yukihiko Sonoda,Teiji Tominaga,Yoko Aoki,Shigeo Kure +18 more
TL;DR: A genodermatosis syndrome associated with BRAF c.1799T>A p.V600E mosaicism is presented, which may represent a new entity in the mosaic RASopathies, partly overlapping with Schimmelpenning‐Feuerstein‐Mims syndrome, which is driven by mosaicism of HRAS and/or KRAS activating mutations.
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Consistent focal cerebral ischemia without posterior cerebral artery occlusion and its real-time monitoring in an intraluminal suture model in mice.
TL;DR: Laser speckle flowmetry could predict the territory of infarction and improve the consistency of the infarct size and small changes in the coating length of the intraluminal suture may be critical to produce consistent MCAO without involving the PCA territory.
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Initiation and propagation of lipid peroxidation in cerebral infarction models. Experimental studies.
TL;DR: The possibility that cerebral ischaemia or cerebral hypoxia may initiate a series of free radical reactions in brain lipid constituents was explored by measuring sequential changes in chemiluminescence (CL) and electron spin resonance (ESR) during hypoxic or ischaemic load.
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