Takamasa Ohno
Nagoya City University
7 Papers
122 Citations
Takamasa Ohno is an academic researcher from Nagoya City University. The author has contributed to research in topics: Phellinus linteus & Cytotoxicity. The author has an hindex of 5, co-authored 7 publications. Previous affiliations of Takamasa Ohno include Aichi Gakuin University.
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Papers
Antimetastatic activity of acteoside, a phenylethanoid glycoside.
TL;DR: It is suggested that acteoside showed suppressive effect on lung metastasis of B16 melanoma cells, and prolonged survival time significantly and the average survival time was 63.3 +/- 3.4d compared with 52.1 +/- 2.5d in control mice.
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Inhibition of cell cycle progression through specific phase by pancratistatin derivatives.
TL;DR: It is suggested that of Amaryllidaceae alkaloids, HBP blocks the progression of cell cycle at least at G0/G1 and S phases and GBP does at least in G0-G1 phase, resulting in apoptosis induction in tumor cells.
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Suppressive effect of Shichimotsu-koka-to (Kampo medicine) on pulmonary metastasis of B16 melanoma cells.
TL;DR: It is suggested that macrophage function-modulating activity by SKT appears to underlie its antimetastatic activity, which leads to a decrease in the number of lung metastatic surface nodules and the extension of life span.
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Inhibitory effect of oral intake of natural Phellinus linteus fruit body on growth and pulmonary metastasis of B16/BL6 melanoma
Takamasa Ohno,Takamasa Ohno,Yurika Takahashi,Hiroki Tanabe,Hiroki Tanabe,Hideki Hirayama,Hajime Mizukami,Yukio Ogihara,Makoto Inoue,Makoto Inoue +9 more
TL;DR: The results suggest that the oral intake of nPF powder may be useful in immunochemotherapy for cancer because of its life prolongation through the inhibitory effect on growth and metastasis of cancer.
7
Suppression of Macrophage Function by Substances with a Molecular Weight Lower than 3000 Da in B16 Melanoma-Conditioned Medium
TL;DR: The results suggest that metastatic B16 melanoma cells produce two distinct substances: to suppress NO production by macrophages and to kill macrophage-like cell lines and to metastasize to target organs.