Tak Mao Chan
University of Hong Kong
401 Papers
3K Citations
Tak Mao Chan is an academic researcher from University of Hong Kong. The author has contributed to research in topics: Medicine & Lupus nephritis. The author has an hindex of 59, co-authored 388 publications. Previous affiliations of Tak Mao Chan include Pamela Youde Nethersole Eastern Hospital & Queen Mary Hospital.
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Papers
Fungal peritonitis--current status 1998.
TL;DR: Its occurrence rate basically follows the peritonitis rate, and the incidence does not seem to vary among different connection systems (6).
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Effect of mycophenolate mofetil on nitric oxide production and inducible nitric oxide synthase gene expression during renal ischaemia‐reperfusion injury
Sing Leung Lui,Loretta Y.Y. Chan,Xiao Hui Zhang,Wen Zhu,Tak Mao Chan,Peter Chin Wan Fung,Kar Neng Lai +6 more
TL;DR: It is concluded that pre-treatment with MMF inhibits the production of NO and the induction of iNOS gene expression in the kidney during IRI, suggesting that MMF might have the potential to ameliorate renal IRI.
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Clinical course and outcomes of single-organism enterococcus peritonitis in peritoneal dialysis patients
Terence Yip,Kai Chung Tse,Flora S.K. Ng,Ivan Hung,Man Fai Lam,Sydney C.W. Tang,Sing L. Lui,Kar N. Lai,Tak Mao Chan,Wai Kei Lo +9 more
TL;DR: Recent use of antibiotics was a risk factor for the development of ampicillin-resistant Enterococcus peritonitis, in turn possibly predisposing the patients to infections caused by vancomycin-resistant organisms.
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Resolution of eosinophilic peritonitis with Ketotifen
TL;DR: A patient with steroid-resistant eosinophilic peritonitis and its successful resolution with ketotifen is reported, and a short course of intraperitoneal hydrocortisone has been shown to be effective in clearing up the eOSinophilia.
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Binding of anti-dsDNA antibodies to proximal tubular epithelial cells contributes to renal tubulointerstitial inflammation
Susan Yung,CY Ng,Kin Yi Au,Kwok Fan Cheung,Qing Zhang,Chenzhu Zhang,Desmond Y H Yap,Mel K. M. Chau,Tak Mao Chan +8 more
TL;DR: The data demonstrate that anti-dsDNA Abs contribute to inflammatory processes in the tubulointerstitium in lupus nephritis through their binding to proximal renal tubular epithelial cells and induction of pro-inflammatory mediators, and MPA ameliorates anti-DSDNA Ab induced IL-6 and IL-8 secretion in these cells.
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