Sun Ah Park
Ajou University
89 Papers
475 Citations
Sun Ah Park is an academic researcher from Ajou University. The author has contributed to research in topics: Dementia & Medicine. The author has an hindex of 23, co-authored 83 publications. Previous affiliations of Sun Ah Park include Soonchunhyang University & Yonsei University.
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Papers
Insulin-degrading enzyme secretion from astrocytes is mediated by an autophagy-based unconventional secretory pathway in Alzheimer disease
Sung Min Son,Moon Yong Cha,Heesun Choi,Seokjo Kang,Hyunjung Choi,Myung-Shik Lee,Sun Ah Park,Inhee Mook-Jung +7 more
TL;DR: It is found that IDE levels were reduced in the cerebrospinal fluid of patients with AD and in pathology-bearing AD-model mice, and that the regulation of autophagy is a potential therapeutic target in addressing Aβ pathology.
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Gender differences in risk factors for transition from mild cognitive impairment to Alzheimer's disease: A CREDOS study.
Sangha Kim,Min-Ji Kim,Seonwoo Kim,Hyo Kang,Shin Won Lim,Woojae Myung,Yunhwan Lee,Chang Hyung Hong,Seong Hye Choi,Duk L. Na,Sang Won Seo,Bon D. Ku,Seong Yoon Kim,SangYun Kim,Jee Hyang Jeong,Sun Ah Park,Bernard J. Carroll,Doh Kwan Kim +17 more
TL;DR: Risk factors for progression from MCI to probable AD differed in men and women, and these results may translate to gender-specific preventative or therapeutic strategies for patients with MCI.
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Cisplatin-induced apoptotic cell death in mouse hybrid neurons is blocked by antioxidants through suppression of cisplatin-mediated accumulation of p53 but not of Fas/Fas ligand.
TL;DR: The results suggest that the neuroprotective effects of antioxidants against cisplatin‐induced neurotoxicity in hybrid neurons are mediated mainly through the inhibition of p53 accumulation but not of Fas/Fas‐L accumulation by these antioxidants.
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Tau mis-splicing in the pathogenesis of neurodegenerative disorders.
TL;DR: A more comprehensive understanding of the relationship between tau mis-splicing and neurodegenerative disorders will aid in the development of efficient therapeutic strategies for patients with a tauopathy or other, related neurodegnerative disorders.
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Simultaneous mitochondrial Ca2+ overload and proteasomal inhibition are responsible for the induction of paraptosis in malignant breast cancer cells
TL;DR: It is found that simultaneous inhibition of the mitochondrial Na(+)/Ca(2+) exchanger (mNCX) and proteasomes can trigger a sustained mitochondrial Ca( 2+) overload and effectively induce paraptosis in malignant breast cancer cells.
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