Stanley H. Appel
Houston Methodist Hospital
452 Papers
6.3K Citations
Stanley H. Appel is an academic researcher from Houston Methodist Hospital. The author has contributed to research in topics: Amyotrophic lateral sclerosis & Medicine. The author has an hindex of 97, co-authored 443 publications. Previous affiliations of Stanley H. Appel include University of Pennsylvania & Baylor University.
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Papers
Patent
Assay for neuromuscular diseases
Ira L. Goldknopf,Essam A. Sheta,Stanley H. Appel,Jennifer K. Bryson,Lemuel Moye,Albert A. Yen,Brian Folsom,Miguel Mosqueda +7 more
- 26 Apr 2006
TL;DR: In this paper, the authors present an assay for determining if a patient has a neuromuscular disease. But, their method is based on the statistical analysis of the results of the test.
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Interaction of myasthenic immunoglobulins and cholinergic agonists on acetylcholine receptors of rat myotubes.
TL;DR: Results with this in vitro model suggest that chronic anticholinesterase therapy in the presence of MG AChR antibodies may aggravate failure of neuromuscular transmission in MG.
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Patent
TRIM 5 related protein as a biomarker of neurodegenerative disease
Ira L. Goldknopf,Essam A. Sheta,Stanley H. Appel,Ericka Simpson,Albert A. Yen +4 more
- 19 Jan 2005
TL;DR: In this paper, a tripartite motif protein TRIM5 isoform gamma related protein was identified as a biomarker useful for the detection, diagnosis, and differentiation of neurodegenerative disease, including but not limited to ALS, AD, and Parkinson's.
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Multimodal 18F-AV-1451 and MRI Findings in Nonfluent Variant of Primary Progressive Aphasia: Possible Insights on Nodal Propagation of Tau Protein Across the Syntactic Network.
Belen Pascual,Quentin Funk,Paolo Zanotti-Fregonara,Neha Pal,Elijah Rockers,Meixiang Max Yu,Bryan Spann,Gustavo C. Román,Paul E. Schulz,Christof Karmonik,Stanley H. Appel,Joseph C. Masdeu +11 more
TL;DR: Tau accumulation likely started in the more affected anterior node and appeared as well in the brain region (in the temporal lobe) spatially separate from but most connected with it and support the idea that neurons that wire together die together.
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Patent
2'-5'-oligoadenylate synthetase like protein as a biomarker for neurodegenerative disease
Ira L. Goldknopf,Essam A. Sheta,Stanley H. Appel,Ericka Simpson,Albert A. Yen +4 more
- 19 Jan 2005
TL;DR: In this article, the identification of an 2′-5′-oligoadenylate synthetase like protein (p59 OASL) as a biomarker useful for the detection, diagnosis, and differentiation of neurodegenerative disease, including but not limited to ALS, AD, and Parkinson's (PD) was discussed.
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