Siwen Li
Northeast Forestry University
30 Papers
42 Citations
Siwen Li is an academic researcher from Northeast Forestry University. The author has contributed to research in topics: Medicine & Chemistry. The author has an hindex of 9, co-authored 16 publications.
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Papers
Subchronic arsenism-induced oxidative stress and inflammation contribute to apoptosis through mitochondrial and death receptor dependent pathways in chicken immune organs
TL;DR: It is demonstrated that the activation of mitochondria and death receptor apoptosis pathways can lead to excessive apoptosis in immune organs of chickens, which suffer from subchronic arsenism, meanwhile, oxidative stress as well as subsequent inflammatory is a crucial driver of apoptosis.
The inflammatory responses in Cu-mediated elemental imbalance is associated with mitochondrial fission and intrinsic apoptosis in Gallus gallus heart.
TL;DR: It is speculated that Cu-induced redistribution of trace elements contributed to inflammatory response and disrupted the mitochondrial network via fission and intrinsic apoptosis in the heart of chickens.
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Endogenous hydrogen sulfide counteracts polystyrene nanoplastics-induced mitochondrial apoptosis and excessive autophagy via regulating Nrf2 and PGC-1α signaling pathway in mouse spermatocyte-derived GC-2spd(ts) cells.
TL;DR: In this paper , the authors investigated the mechanisms of NaPs-induced reproductive toxicity in vitro and probable reproductive protection by endogenous hydrogen sulfide (H2S) in GC-2spd(ts) cells.
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Regulation of autophagy factors by oxidative stress and cardiac enzymes imbalance during arsenic or/and copper induced cardiotoxicity in Gallus gallus.
TL;DR: As or/and Cu induced cardiac damage and autophagy via elevating cardiac enzymes activities, inducing oxidative stress and activating the PI3K/AKT/mTORC pathway in heart of chickens.
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NF-κB-mediated inflammation correlates with calcium overload under arsenic trioxide-induced myocardial damage in Gallus gallus.
TL;DR: It is indicated that As2O3-induced inflammatory response might dependent on Ca overload in myocardial damage of chickens, and has implications for the development of potential therapeutic approaches by resisting Ca overload for arsenic-induced myocardIAL damage.
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