Shuai Wang
15 Papers
Shuai Wang is an academic researcher. The author has contributed to research in topics: Medicine & Chemistry. The author has an hindex of 2, co-authored 10 publications.
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Papers
Analysis of the absorbed constituents and mechanism of liquidambaris fructus extract on hepatocellular carcinoma
TL;DR: The pharmacodynamic material basis of LFE is revealed and from the perspective of metabolomics proved that the effects of inhibiting the growth of tumor cells, promoting tumor cell apoptosis, reducing inflammatory reaction, protecting hepatocytes, improving the survival state of tumor rats, and prolonging the life cycle are related to its impact on PTEN/PI3K/Akt, fatty acid metabolism and other key signal pathways.
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Possible mechanisms associated with immune escape and apoptosis on anti-hepatocellular carcinoma effect of Mu Ji Fang granules
Yi-bing Zhang,Yong-Rui Bao,Shuai Wang,Tianjiao Li,H. Tai,Jianjian Leng,Xin-Xin Yang,Bo Wang,Xian-Sheng Meng +8 more
TL;DR: In this paper , the main ingredients in Mu Ji Fang granules (MJF) are alkaloids, flavonoids, and polysaccharides, which are used in the clinical treatment of hepatitis, cirrhosis and HCC for more than 30 years.
1
Classical prescription Floris Sophorae Powder treat colorectal cancer by regulating KRAS/MEK-ERK signaling pathway.
Li-Ying Han,Hao Yu,Shuai Wang,Yong-Rui Bao,Tianjiao Li,Ying Zheng,Xinglan Luo,Meng-Nan Jia,Qiang Zhang,Xian-Sheng Meng +9 more
TL;DR: This study investigates the efficacy of classical prescription Floris Sophorae Powder in treating colorectal cancer by regulating the KRAS/MEK-ERK signaling pathway, demonstrating its potential as a therapeutic agent through in vivo experiments and serum metabolomics analysis.
1
Effect of Oroxylum indicum on hepatocellular carcinoma via the P53 and VEGF pathways based on microfluidic chips
Xinglan Luo,Miao Zhao,Sicong Liu,Ying Zheng,Qiang Zhang,Yong-Rui Bao,Shuai Wang,Tianjiao Li,Xian-Sheng Meng +8 more
TL;DR: The findings suggest that OI may inhibit the migration, invasion, and neovascularization of HepG2 cells, and its regulatory mechanism may be related to the regulation of the P53 and VEGF pathways.
1