Shan Jiang
Nankai University
5 Papers
9 Citations
Shan Jiang is an academic researcher from Nankai University. The author has contributed to research in topics: Cancer & Tumor microenvironment. The author has an hindex of 5, co-authored 5 publications. Previous affiliations of Shan Jiang include Xi'an Jiaotong University.
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Papers
STIM1 accelerates cell senescence in a remodeled microenvironment but enhances the epithelial-to-mesenchymal transition in prostate cancer.
Yingxi Xu,Shu Zhang,Haiying Niu,Yujie Ye,Fen Hu,Si Chen,Xuefei Li,Xiaohe Luo,Shan Jiang,Yanhua Liu,Yanan Chen,Junying Li,Rong Xiang,Na Li +13 more
TL;DR: This study revealed novel regulatory effects and the mechanisms by which STIM1 affects cell senescence, tumor migration and the tumor microenvironment, revealing that STIM 1 has multiple functions in prostate cancer cells.
Phosphodiesterase 3A Represents a Therapeutic Target that Drives Stem Cell-like Property and Metastasis in Breast Cancer.
Na Hao,Wenzhi Shen,Wenzhi Shen,Wenzhi Shen,Renle Du,Shan Jiang,Junyong Zhu,Yanan Chen,Yanan Chen,Chongbiao Huang,Yi Shi,Yi Shi,Rong Xiang,Rong Xiang,Yunping Luo +14 more
TL;DR: It is shown that PDE3A could predispose patients with breast cancer to metastases by acting as a mediator of cancer stemness and is a potential therapeutic target for advanced breast cancer.
TLR4 increases the stemness and is highly expressed in relapsed human hepatocellular carcinoma
Shuang Zhou,Renle Du,Zhenglu Wang,Wenzhi Shen,Ruifang Gao,Shan Jiang,Yan Fang,Yuzhi Shi,Antao Chang,Lei Liu,Chenghu Liu,Na Li,Rong Xiang +12 more
TL;DR: The discovery that the expression of TLR4 in relapsed human hepatocellular carcinoma clinical samples is higher than that in the non‐relapsed ones provides a novel therapeutic target to prevent the recurrence of HCC.
25
•Journal Article
Role of Hyaluronan and Glucose on 4-Methylumbelliferone-inhibited Cell Proliferation in Breast Carcinoma Cells
Rongrong Wang,Wei Zhou,Juan Wang,Yanhua Liu,Yanan Chen,Shan Jiang,Xiaohe Luo,Dwayne G. Stupack,Na Luo +8 more
TL;DR: Increased amounts of hyaluronan or glucose in the tumor microenvironment reduced cellular sensitivity of breast carcinoma cells to 4-MU-inhibited cell proliferation.
11
ICAM3 mediates inflammatory signaling to promote cancer cell stemness.
Wenzhi Shen,Junling Xie,Shuangtao Zhao,Renle Du,Xiaohe Luo,Huiwen He,Shan Jiang,Na Hao,Chong Chen,Chunlei Guo,Yanhua Liu,Yanan Chen,Peiqing Sun,Shengyong Yang,Na Luo,Rong Xiang,Yunping Luo +16 more
TL;DR: A potential CSC regulator is identified and a novel mechanism by which ICAM3 governs cancer cell stemness and inflammation is suggested, which results in tumor growth suppression in vitro and in–vivo.