Selvi Celik
Lund University
16 Papers
3 Citations
Selvi Celik is an academic researcher from Lund University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 7, co-authored 9 publications.
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Papers
Exosomal miR-142-3p is increased during cardiac allograft rejection and augments vascular permeability through down-regulation of endothelial RAB11FIP2 expression
Ihdina Sukma Dewi,Selvi Celik,Anna Karlsson,Zsuzsanna Hollander,Karen K. Lam,Janet Wilson McManus,Scott J. Tebbutt,Raymond T. Ng,Paul Keown,Robert McMaster,Bruce M. McManus,Jenny Öhman,Olof Gidlöf +12 more
TL;DR: A novel mechanism whereby miR-142-3p, a microRNA enriched in exosomes during acute cellular rejection, is transferred to endothelial cells and compromises endothelial barrier function via down-regulation of RAB11FIP2 is identified.
Treatment with a GnRH receptor agonist, but not the GnRH receptor antagonist degarelix, induces atherosclerotic plaque instability in ApoE-/- mice
Anki Knutsson,Sabrina Hsiung,Selvi Celik,Sara Rattik,Ingrid Yao Mattisson,Maria Wigren,Howard I. Scher,Jan Nilsson,Anna Hultgårdh-Nilsson +8 more
TL;DR: Destabilization of pre-existing atherosclerotic plaques could explain the increased cardiovascular risk in prostate cancer patients treated with GnRH-R agonists.
Inhibition of the long non-coding RNA NEAT1 protects cardiomyocytes from hypoxia in vitro via decreased pri-miRNA processing.
Olof Gidlöf,Kerstin Bader,Selvi Celik,Mario Grossi,Shinichi Nakagawa,Tetsuro Hirose,Bernhard Metzler,Björn Olde,David Erlinge +8 more
TL;DR: The nuclear lncRNA Neat1 is identified as part of a conserved oxygen-sensitive feedback mechanism by regulation of miRNA processing and a potential target in cardioprotection.
Functional Screening Identifies MicroRNA Regulators of Corin Activity and Atrial Natriuretic Peptide Biogenesis
TL;DR: A novel regulator of corin activity and ANP biogenesis in human cardiomyocytes that might be of potential future therapeutic utility is identified.
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Vascular smooth muscle-specific YAP/TAZ deletion triggers aneurysm development in mouse aorta.
Marycarmen Arévalo Martinez,Olivia Ritsvall,Joakim Bastrup,Selvi Celik,G. Jakobsson,Fatima Daoud,Christopher Winqvist,Anders Aspberg,Catarina Rippe,Lars Maegdefessel,Alexandru Schiopu,Thomas A. Jepps,Johan Holmberg,Karl Swärd,Sebastian Albinsson +14 more
TL;DR: Reduced expression of YAP1 in human aortic aneurysms is identified using the integrin α8–Cre (Itga8-Cre) mouse model, which represents a rapid and spontaneousAneurysm model that recapitulates features of human abdominal aorta and immune cell populations.
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