Scott D. Smid
University of Adelaide
56 Papers
380 Citations
Scott D. Smid is an academic researcher from University of Adelaide. The author has contributed to research in topics: Chemistry & Cannabinoid. The author has an hindex of 19, co-authored 51 publications. Previous affiliations of Scott D. Smid include Royal Adelaide Hospital & Bond University.
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Papers
Cannabinoid receptor agonism inhibits transient lower esophageal sphincter relaxations and reflux in dogs
Anders Lehmann,L. Ashley Blackshaw,Lena Brändén,Anita Carlsson,Jörgen Jensen,Emelie Nygren,Scott D. Smid +6 more
TL;DR: Exogenous and endogenous activation of theCBR1 receptor inhibits TLESRs, and the effects of CBR1 are not mediated peripherally on gastric vagal afferents, and therefore are most likely in the brain stem.
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In vitro studies of the neuroprotective activities of astaxanthin and fucoxanthin against amyloid beta (Aβ1-42) toxicity and aggregation.
TL;DR: Astaxanthin and fucoxanthin provided neuroprotection against A&bgr;1–42 and both compounds demonstrated multiple neuroprotective activities, including anti‐aggregation effects and neurite outgrowth activity.
104
Phlorotannins: A review on biosynthesis, chemistry and bioactivity
TL;DR: A library of reported phlorotannins has been generated to assist with further identification, and the majority of such findings have been generated via biochemical and cell-based assays, with only a limited number of in vivo animal experiments conducted.
100
Cannabinoid 1 (CB1) receptors coupled to cholinergic motorneurones inhibit neurogenic circular muscle contractility in the human colon
TL;DR: In conclusion, activation of CB1 receptors coupled to cholinergic motorneurones selectively and reversibly inhibits excitatory nerve transmission in colonic human colonic CM.
GABABR expressed on vagal afferent neurones inhibit gastric mechanosensitivity in ferret proximal stomach
TL;DR: GABA(B)R expressed on vagal afferent fibers directly inhibit gastric mechanosensory activity, likely a contributing mechanism to the efficacy of GABA(B)-receptor agonists in reducing TLESR and reflux episodes in vivo.
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