Scott B. Evans

TL;DR: Results indicate that the CNS roles of insulin and leptin may include processes involving memory and reward, and elevated insulin andptin do not prevent learning a food's reward value, but instead block its retrieval.

TL;DR: It is concluded that NMDARs in dopamine neurons enhance but are not critical for phasic dopamine release to behaviorally relevant stimuli; furthermore, their contribution to phAsic dopamine signaling is not necessary for the development of cue-evoked dopamine or anticipatory activity in a D1R-dependent Pavlovian conditioning paradigm.

TL;DR: Results suggest that decreased activation of the PVN may be important in the development of HAAF and that antecedent exposure to elevated levels of Cort is not always sufficient to produce HAAF.

TL;DR: It appears that decreased PVN activation, as occurs with HAAF, may be involved in specific components of HAAF (i.e., blunting the sympathoadrenal and hypothalamic-pituitary-adrenocortical axis response), but not in others.