Satoshi Sumida
Osaka Gakuin University
11 Papers
107 Citations
Satoshi Sumida is an academic researcher from Osaka Gakuin University. The author has contributed to research in topics: Internal medicine & Incremental exercise. The author has an hindex of 8, co-authored 11 publications.
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Papers
Effect of repeated exercise on urinary 8-hydroxy-deoxyguanosine excretion in humans.
Koji Okamura,Tatsuya Doi,Koichiro Hamada,Masao Sakurai,Yasuyuki Yoshioka,Ryoichi Mitsuzono,Takashi Migita,Satoshi Sumida,Yohko Sugawa-Katayama +8 more
TL;DR: The results indicate that repeated exercise augments oxidative stress and the DNA is also injured by exercise-induced reactive oxygen species.
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Effect of a single bout of exercise and beta -carotene supplementation on the urinary excretion of 8-hydroxy-deoxyguanosine in humans
TL;DR: The results suggest that a single bout of incremental exercise does not induce the oxidative DNA damage, while beta-carotene supplementation may attenuate it.
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Transient responses in cardiac function below, at, and above anaerobic threshold.
Kiyoji Tanaka,Takayoshi Yoshimura,Satoshi Sumida,Ryoichi Mitsuzono,Shinsuke Tanaka,Yotaro Konishi,Hitoshi Watanabe,Takashi Yamada,Kazuya Maeda +8 more
TL;DR: Stroke volume (SV) and stroke index (SI) at rest and during submaximal and maximal exercise, determined by impedance cardiography, were consistently greater in the trained than in the less fit group.
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No influence of a single bout of exercise on urinary excretion of 8-hydroxy-deoxyguanosine in humans
Satoshi Sumida,Koji Okamura,Tatsuya Doi,Masao Sakurai,Yasuyuki Yoshioka,Yohko Sugawa-Katayama +5 more
TL;DR: The results suggest that the oxidative stress during a single bout of intensive exercise does not result in an accumulation of oxidative DNA damage.
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Effect of Endurance Exercise on the Tissue 8-Hydroxy-deoxyguanosine Content in Dogs
Koji Okamura,Tatswa Doi,Masao Sakurai,Koichiro Hamada,Yasuyuki Yoshioka,Satoshi Sumida,Yohko Sugawa-Katayama +6 more
TL;DR: Results indicate that, immediately after endurance exercise, an augmented repair mechanism might thus play a role in the decrease of 8-OHdG in the lymphocytes and the colon, while the 8- OHdG generation might be counterbalanced by its repair in other tissues.
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