Samuel Solomon
University of Konstanz
12 Papers
19 Citations
Samuel Solomon is an academic researcher from University of Konstanz. The author has contributed to research in topics: Arthritis & Complement system. The author has an hindex of 10, co-authored 12 publications. Previous affiliations of Samuel Solomon include University of British Columbia.
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Papers
Targeted mast cell silencing protects against joint destruction and angiogenesis in experimental arthritis in mice
Manfred Kneilling,Lothar Hültner,Bernd J. Pichler,Reinhard Mailhammer,Lars Morawietz,Samuel Solomon,Martin Eichner,Joseph Sabatino,Tilo Biedermann,Veit Krenn,Wolfgang A. Weber,Harald Illges,Roland Haubner,Martin Röcken +13 more
TL;DR: It is shown that intraarticular and intraperitoneal mast cell engraftment fully restores susceptibility to antibody-induced arthritis, angiogenesis, and alphavbeta3 integrin activation in GPI antibody- induced arthritis.
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Fragile mental retardation protein interacts with the RNA-binding protein Caprin1 in neuronal ribonucleoprotein complexes
Rachid El Fatimy,Sandra Tremblay,Alain Y. Dury,Samuel Solomon,Paul De Koninck,John W. Schrader,Edouard W. Khandjian +6 more
TL;DR: In vivo and in vitro evidence show that Caprin1 interacts with FMRP at the level of the translation machinery as well as in trafficking neuronal granules, and it is proposed that protein interactors might modulate FMRp functions.
Transmission of antibody-induced arthritis is independent of complement component 4 (C4) and the complement receptors 1 and 2 (CD21/35).
Samuel Solomon,Cornelia Kolb,Subhasis Mohanty,Elvira Jeisy-Walder,Rosemarie Preyer,Volkmar Schöllhorn,Harald Illges +6 more
TL;DR: The transmission of K/BxN serum‐induced RA is independent of the classical as well as the lectin complement pathways and CR1 and CR2, and the crucial role of complement C5, while neither classical nor lectin pathway is necessary, indicates that the alternative complement pathway may have a role in the K/ Bxn serum‐ induced RA model.
50
Histidine decarboxylase but not histamine receptor 1 or 2 deficiency protects from K/BxN serum-induced arthritis
Narendiran Rajasekaran,Samuel Solomon,Takeshi Watanabe,Hiroshi Ohtsu,Mieczyslaw Gajda,Rolf Bräuer,Harald Illges +6 more
TL;DR: It is indicated that histamine is required but not indispensable for the development of serum-induced arthritis and histamine receptors other than those studied here may be involved.