Sachiko Miyagawa-Tomita
University of Tokyo
61 Papers
503 Citations
Sachiko Miyagawa-Tomita is an academic researcher from University of Tokyo. The author has contributed to research in topics: Neural crest & Medicine. The author has an hindex of 28, co-authored 54 publications. Previous affiliations of Sachiko Miyagawa-Tomita include Yamazaki Gakuen University.
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Papers
Cardiac neural crest cells provide new insight into septation of the cardiac outflow tract: aortic sac to ventricular septal closure.
TL;DR: The present study describes the movement of cardiac neural crest cells from the caudal pharyngeal arches into the outflow tract and base of the heart during the period of outflow septation and finds that not all of the condensed mesenchyme in the out flow tract during septations was derived from neural crest.
296
First Evidence That Bone Marrow Cells Contribute to the Construction of Tissue-Engineered Vascular Autografts In Vivo
TL;DR: These TEVAs are useful for cardiovascular surgery in humans and especially in children, who require biocompatible materials with growth potential, which might reduce the instance of complications caused by incompatible materials and lead to a reduced likelihood of further surgery.
292
Mesp1 Expression Is the Earliest Sign of Cardiovascular Development
TL;DR: A transcription factor, Mesp1, is expressed in almost all precursors of the cardiovascular system and plays an essential role in cardiac morphogenesis, and may play a key role in the early specification for cardiac precursor cells.
263
Active Remodeling of the Coronary Arterial Lesions in the Late Phase of Kawasaki Disease Immunohistochemical Study
Atsuko Suzuki,Sachiko Miyagawa-Tomita,Keiko Komatsu,Toshio Nishikawa,Yasunari Sakomura,Toshinobu Horie,Makoto Nakazawa +6 more
TL;DR: In this article, the authors examined formalin-fixed specimens of the coronary arteries immunohistochemically by using antibodies against vascular growth factors (GFs) and their receptors in 7 children with Kawasaki disease, 9 children with no coronary disease, and 3 adults with atherosclerosis.
193
Mouse hesr1 and hesr2 genes are redundantly required to mediate Notch signaling in the developing cardiovascular system.
Hiroki Kokubo,Hiroki Kokubo,Sachiko Miyagawa-Tomita,Makoto Nakazawa,Yumiko Saga,Yumiko Saga,Randy L. Johnson +6 more
TL;DR: It is reported that mice lacking the Hesr1 gene are viable and fertile, whereas knockout mouse of both hesr1 and hesr2 is embryonic lethal at 11.5 days postcoitum and recapitulates most of the known cardiovascular phenotypes of disrupted Notch pathway mutants including defects in arterial-venous specification, septation and cushion formation.
189