Ryuji Shingaki
Okayama University
8 Papers
89 Citations
Ryuji Shingaki is an academic researcher from Okayama University. The author has contributed to research in topics: Swarming motility & Bacillus subtilis. The author has an hindex of 6, co-authored 8 publications.
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Papers
Genome-Wide Screening of Genes Required for Swarming Motility in Escherichia coli K-12
TL;DR: Lipopolysaccharide and the enterobacterial common antigen may be important surface-acting components that contribute to the reduction of surface tension, thereby facilitating the swarm migration in the E. coli K-12 strain.
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Inhibition of swarming motility of Pseudomonas aeruginosa by branched-chain fatty acids
TL;DR: A branched-chain fatty acid (BCFA)--12-methyltetradecanoic acid (anteiso-C15:0)--has slightly repressed flagella-driven swimming motility and completely inhibited a more complex type of surface motility, i.e. swarming, at a concentration of 10 microg mL(-1).
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Biofilm formation by a fimbriae-deficient mutant of Actinobacillus actinomycetemcomitans.
Tetsuyoshi Inoue,Ryuji Shingaki,Norio Sogawa,Chiharu Sogawa,Jun Ichi Asaumi,Susumu Kokeguchi,Kazuhiro Fukui +6 more
TL;DR: Findings suggest that fimbriae are not essential for the surface‐adherent growth but are required for enhancing cell‐to‐surface and cell‐ to‐cell interactions to stabilize the biofilm.
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Heterogeneous Post-Translational Modification of Actinobacillus actinomycetemcomitans Fimbrillin
TL;DR: The exact molecular mass of A. actinomycetemcomitans fimbrillin, a structural subunit of fimbriae, was determined by liquid chromatography‐electrospray ionization mass spectrometry and suggested that the fimbrial peptides were post‐translationally modified.
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Chromosome DNA fragmentation and excretion caused by defective prophage gene expression in the early-exponential-phase culture of Bacillus subtilis.
TL;DR: The present findings suggest that the spontaneous generation and excretion of chromosome DNA fragments in Bacillus subtilis are both closely related to the expression of defective prophage genes.
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