Rui Li
Anhui Medical University
30 Papers
107 Citations
Rui Li is an academic researcher from Anhui Medical University. The author has contributed to research in topics: Medicine & Odds ratio. The author has an hindex of 10, co-authored 23 publications.
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Papers
Role of interleukin-10 and interleukin-10 receptor in systemic lupus erythematosus
TL;DR: A better understanding of the regulation of IL-10 and its receptors (IL-10R) can likely provide more valuable clues to the pathogenic mechanisms underlying specific forms of SLE, so as to pave the way toward more effective therapeutics.
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Prevalence of systemic lupus erythematosus and risk factors in rural areas of Anhui Province
Yan-Feng Zou,Chen-Chen Feng,Ji-Min Zhu,Ji-Min Zhu,Jin-Hui Tao,Gui-Mei Chen,Qian-Ling Ye,Han Cen,Rui-Xue Leng,Faming Pan,Hai-Feng Pan,Rui Li,Yin-Guang Fan,Bin Wang,Xiang-Pei Li,Feng-Yu Zhang,Dong-Qing Ye +16 more
TL;DR: A large population-based epidemiological survey explored the prevalence of SLE and risk factors in rural areas of Anhui Province of China and estimated the prevalence at 37.56 per 100,000 persons.
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Gene-gene and gene-sex epistatic interactions of MiR146a, IRF5, IKZF1, ETS1 and IL21 in systemic lupus erythematosus.
Rui-Xue Leng,Wei Wang,Han Cen,Mo Zhou,Chen-Chen Feng,Yan Zhu,Xiao-Ke Yang,Mei Yang,Yu Zhai,Bao-Zhu Li,Xiao-Song Wang,Rui Li,Gui-Mei Chen,Hong Chen,Hai-Feng Pan,Dong-Qing Ye +15 more
TL;DR: This study identified novel gene–gene/gene-sex interactions in lupus and highlights sex, interferon pathway, and Th17/B cells as important contributors to the pathogenesis of SLE.
Association of P2X7R gene polymorphisms with systemic lupus erythematosus in a Chinese population
Gui-Mei Chen,Chen-Chen Feng,Qian-Ling Ye,Tao Jin-hui,Rui Li,Hui Peng,Mo Zhou,Rui-Xue Leng,Jing Li,Han Cen,Yin-Guang Fan,Hai-Feng Pan,Dong-Qing Ye +12 more
TL;DR: The findings suggest that the P2X7R gene might contribute to SLE susceptibility in the Chinese population, and one haplotype appeared to be a significantly 'protective' haplotype.
METTL3 boosts glycolysis and cardiac fibroblast proliferation by increasing AR methylation.
Yang Zhou,Kai Song,Bin Tu,He Sun,Jiqiang Ding,Yong Luo,Ji-ming Sha,Rui Li,Ye Zhang,Jian-Yuan Zhao,Hui Tao +10 more
TL;DR: In this article , the authors investigated the cause and consequence of glycolysis reprogramming in cardiac fibrosis, using clinical samples, animal models, and cultured cells, and found that methyltransferase-like 3 (METTL3) facilitates the development of cardiac fibroblasts.
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