Rossella Iori
University of Modena and Reggio Emilia
18 Papers
442 Citations
Rossella Iori is an academic researcher from University of Modena and Reggio Emilia. The author has contributed to research in topics: Bile acid & Cholesterol. The author has an hindex of 13, co-authored 18 publications.
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Papers
Hepatic cholesterol and bile acid metabolism in subjects with gallstones: comparative effects of short erm feeding of chenodeoxycholic and ursodeoxycholic acid.
TL;DR: The results would suggest that in the liver of patients with gallstones the conversion of cholesterol to bile acids is somewhat reduced, and that changing the bile acid pool composition, by exogenous bile Acid feeding, has disparate effects on hepatic cholesterol synthesis.
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Cholesterol absorption during bile acid feeding. Effect of ursodeoxycholic acid (UDCA) administration.
TL;DR: Bile saturation critically depended on the criteria adopted to calculate the saturation index, and UDCA was virtually undetectable in bile before treatment, but became the most abundant bile acid after treatment.
101
The effect of chenodeoxycholic acid (CDCA) on cholesterol absorption.
TL;DR: The effect of administration of chenodeoxycholic acid (CDCA) on cholesterol absorption has been investigated in 11 volunteers and cholesterol saturation of bile and biliary bile acid composition were studied.
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Influence of Small-Bowel Transit Time on Dietary Cholesterol Absorption in Human Beings
TL;DR: It is shown that in certain guinea pigs that are resistant to the pathologic effect of dietary cholesterol, intestinal transit time is more rapid than in other guinea pig that become hypercholesterolemic, suggesting that intestinal transitTime allows less absorption of cholesterol in the former group, thus limiting cholesterol accumulation in body tissues.
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Bile acid feeding and hepatic sterol metabolism: Effect of deoxycholic acid
TL;DR: The effect of deoxycholic acid feeding on hepatic sterol metabolism was studied in 9 subjects with cholesterol gallstones and in the treated group hepatic HMG-CoA reductase was reduced by 45%, as compared with the control value.
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