Roser Pagan
University of Barcelona
17 Papers
256 Citations
Roser Pagan is an academic researcher from University of Barcelona. The author has contributed to research in topics: Vimentin & Cellular differentiation. The author has an hindex of 14, co-authored 17 publications.
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Papers
RGD peptides and monoclonal antibodies, antagonists of alpha(v)-integrin, enter the cells by independent endocytic pathways.
Susanna Castel,Roser Pagan,Francesc Mitjans,Jaume Piulats,Simon L. Goodman,Alfred Jonczyk,Florian Huber,Senén Vilaró,Manuel Reina +8 more
TL;DR: It is proved that melanoma cells pretreated with the mAb do not readhere to the substrate, whereas cellspretreated with cRGD peptide retain their readhesion capacity.
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Fibronectin regulates morphology, cell organization and gene expression of rat fetal hepatocytes in primary culture.
Aránzazu Sánchez,Alberto Alvarez,Roser Pagan,Cesar Roncero,Senén Vilaró,Manuel Benito,Isabel Fabregat +6 more
TL;DR: The results suggest that fibronectin might play a differential role, as compared to other extracellular matrix proteins, in fetal hepatocyte organization and gene expression.
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Epithelial-mesenchymal transition of cultured rat neonatal hepatocytes is differentially regulated in response to epidermal growth factor and dimethyl sulfoxide
TL;DR: Results indicate that EMT of cultured rat neonatal hepatocytes is differentially regulated in response to EGF and DMSO.
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Vimentin filaments follow the preexisting cytokeratin network during epithelial-mesenchymal transition of cultured neonatal rat hepatocytes.
TL;DR: Results are consistent with a mechanism of vimentin assembly, whereby growth of vimentsin intermediate filaments is dependent on microtubules in topographically restricted cytoplasmic sites, in close relation to the cytokeratin cytoskeleton and to changes in cell-cell contact and cell shape.
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Effects of growth and differentiation factors on the epithelial-mesenchymal transition in cultured neonatal rat hepatocytes
TL;DR: Activation of vimentin expression produced in cultures supplemented with the mitogenic factors (EGF and HGF) is independent of the activation of cell growth, because DMSO but not TGF-beta1 can abolish vimentsin synthesis, although both inhibited growth.
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