Robin Looft-Wilson
College of William & Mary
29 Papers
195 Citations
Robin Looft-Wilson is an academic researcher from College of William & Mary. The author has contributed to research in topics: Vasodilation & Enos. The author has an hindex of 15, co-authored 29 publications. Previous affiliations of Robin Looft-Wilson include Ames Research Center & University of Virginia.
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Papers
Endothelial-specific expression of caveolin-1 impairs microvascular permeability and angiogenesis
Philip M. Bauer,Jun Yu,Yan Chen,Reed Hickey,Pascal Bernatchez,Robin Looft-Wilson,Yan Huang,Frank J. Giordano,Radu V. Stan,William C. Sessa +9 more
TL;DR: Data strongly support the idea that, in vivo, Cav-1 may modulate signaling pathways independent of its essential role in caveolae biogenesis, and impairs endothelial nitric oxide synthase activation, endothelial barrier function, and angiogenic responses to exogenous VEGF and tissue ischemia.
175
•Journal Article
Pre-exercise hypervolemia and cycle ergometer endurance in men
John E. Greenleaf,Robin Looft-Wilson,J. L. Wisherd,M. A. McKenzie,Christopher D. Jensen,J. H. Whittam +5 more
TL;DR: The attenuated endurance for N and P1 could not be attributed to differences in exercise metabolism from the carbohydrate or citrate, terminal heart rate, levels of perceived exertion, forehead or thigh skin blood flow velocity, changes or absolute termination levels of rectal temperature.
•Journal Article
Hypervolemia in men from fluid ingestion at rest and during exercise
John E. Greenleaf,Robin Looft-Wilson,J. L. Wisherd,Catherine G. R. Jackson,P. P. Fung,A. C. Ertl,P. R. Barnes,Christopher D. Jensen,J. H. Whittam +8 more
TL;DR: Drink composition appears to be more important than its Osm for increasing PV at rest and for maintaining it during exercise in previously euhydrated subjects.
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Chronic diet-induced hyperhomocysteinemia impairs eNOS regulation in mouse mesenteric arteries
Robin Looft-Wilson,Blair S. Ashley,Janelle Billig,Madeline Wolfert,Lindsay Ambrecht,Shawn E. Bearden +5 more
TL;DR: Chronic diet-induced HHcy in mice impairs eNOS protein expression and phosphorylation at S1179, coincident with impaired NO.-dependent dilation, which implicates dysfunction in eN OS post-transcriptional regulation in the impaired endothelium-dependent vasodilation and microvascular disease that is common with HHcy.
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Independence of connexin expression and vasomotor conduction from sympathetic innervation in hamster feed arteries.
TL;DR: In feed arteries of the hamster retractor muscle, the authors tested whether sympathetic denervation would alter the expression of connexin isoforms and the conduction of vasomotor responses.
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