Robert H. Thalmann
Baylor College of Medicine
13 Papers
428 Citations
Robert H. Thalmann is an academic researcher from Baylor College of Medicine. The author has contributed to research in topics: GABAB receptor & GABAA receptor. The author has an hindex of 12, co-authored 13 publications.
Chat about Author
Papers
Evidence that guanosine triphosphate (GTP)-binding proteins control a synaptic response in brain: effect of pertussis toxin and GTP gamma S on the late inhibitory postsynaptic potential of hippocampal CA3 neurons.
TL;DR: It is proposed that the transmitter receptor of the late IPSP activates a potassium conductance via a G-protein that is sensitive to blockade by pertussis toxin and that GTP gamma S and baclofen activate a conductance that depends upon the same G-proteins and/or potassium channels as does the lateIPSP.
116
Taurine in hippocampus: localization and postsynaptic action.
TL;DR: Results from immunocytochemical studies suggest that taurine may be used as a neurotransmitter only by a small number of pyramidal basket interneurons, and that the vast majority of CSAD-positive neurons may use taurines for other functions.
101
Some factors that influence the decrement in the response to GABA during its continuous iontophoretic application to hippocampal neurons.
TL;DR: This response decrement that occurs during continuous iontophoretic application of GABA to hippocampal neurons was characterized by intracellular methods in the rat hippocampal slice and was appropriate to participation in the development of cellular responses to brief flurries of GABA-mediated inhibitory postsynaptic potentials that may occur normally, or that might occur abnormally during a seizure or artificial tetany.
49
Regulation of γ-aminobutyric acidB (GABAB) receptors in cerebral cortex during the estrous cycle
TL;DR: The robust regulation of GABAB receptors in neocortex was unexpected and its functional significance is at present unknown, however, the correlation of the menstrual cycle with mood and other behavioral changes, and the correlations of the estrous and menstrual cycles with seizure susceptibility, may somehow depend upon hormonal regulation of transmitter systems such as the one observed here.
43
Pertussis toxin blocks a late inhibitory postsynaptic potential in hippocampal CA3 neurons
TL;DR: It is proposed that a pertussis toxin-sensitive G-protein controls the conductance of the late IPSP, an inactivator of several GTP-binding proteins (G-proteins) excluding the G- protein that stimulates adenylyl cyclase.
38