Robert A. Moyer
Ohio State University
6 Papers
2 Citations
Robert A. Moyer is an academic researcher from Ohio State University. The author has contributed to research in topics: Arrestin & Engineering. The author has an hindex of 4, co-authored 5 publications.
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Papers
An Opioid Agonist that Does Not Induce μ-Opioid Receptor—Arrestin Interactions or Receptor Internalization
Chad E. Groer,Kevin Tidgewell,Robert A. Moyer,Wayne W. Harding,Richard B. Rothman,Thomas E. Prisinzano,Laura M. Bohn +6 more
TL;DR: Evaluating μOR trafficking in response to activation by a novel μ-selective agonist derived from the naturally occurring plant product, salvinorin A, finds that this compound, termed herkinorin, does not promote the recruitment of β-arrestin-2 to the μOR and does not lead to receptor internalization.
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Intronic polymorphisms affecting alternative splicing of human dopamine D2 receptor are associated with cocaine abuse.
Robert A. Moyer,Danxin Wang,Audrey C. Papp,Ryan M. Smith,Linda Duque,Deborah C. Mash,Wolfgang Sadee +6 more
TL;DR: The role of rs2283265/rs1076560 in D2 alternative splicing and its effects on reducing formation of D2S relative to D2L were confirmed and support a strong role in susceptibility to cocaine abuse.
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KAPPA OPIOIDS PROMOTE THE PROLIFERATION OF ASTROCYTES VIA Gβγ AND β-ARRESTIN 2 DEPENDENT MAPK-MEDIATED PATHWAYS
Gregory P. McLennan,Alexi Kiss,Mayumi Miyatake,Mariana M. Belcheva,Kari T. Chambers,John J. Pozek,Yasmin Mohabbat,Robert A. Moyer,Laura M. Bohn,Carmine J. Coscia +9 more
TL;DR: Proliferative actions of both agonists were abolished by either inhibition of ERK1/2, Gβγ subunits or β‐arrestin 2, suggesting that both G protein‐dependent and ‐independent ERK pathways are required for this outcome.
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A procedure to detect and identify specific chemicals of potential inhalation toxicity concern in aerosols
Theodore P. Klupinski,Robert A. Moyer,Po-Hsu Allen Chen,Erich D. Strozier,Stephanie S. Buehler,David A. Friedenberg,Bartosz Koszowski +6 more
TL;DR: In this paper , a non-targeted chemical analysis by two-dimensional gas chromatography-time-of-flight mass spectrometry (GC× GC-TOFMS) and assessment of the results using publicly available toxicity data to prioritize the tentatively identified detected chemicals according to potential inhalation toxicity.