Raymond L. Jackson
University of Texas at Arlington
19 Papers
812 Citations
Raymond L. Jackson is an academic researcher from University of Texas at Arlington. The author has contributed to research in topics: Learned helplessness & Shock (circulatory). The author has an hindex of 13, co-authored 19 publications. Previous affiliations of Raymond L. Jackson include University of Colorado Boulder.
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Papers
Long-Term Analgesic Effects of Inescapable Shock and Learned Helplessness
TL;DR: Although exposure to inescapable shocks induced analgesia in rats, the analgesia was not manifest 24 hours later, and a brief reexposure to shock had an analgesic effect only if the rats had been shocked 24 hours previously.
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Opiate antagonists and long-term analgesic reaction induced by inescapable shock in rats.
Steven F. Maier,Susan Davies,James W. Grau,Raymond L. Jackson,Daniel H. Morrison,Thomas B. Moye,John Madden,Jack D. Barchas +7 more
TL;DR: Five experiments examined the influence of opiate antagonists on both the short-term analgesic reaction resulting 30 min after exposure to inescapable shock and the long-term narcotic reaction resulting after reexposure to shock 24 hr after inescapables shock exposure.
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Differential effect of anterior cingulate cortex lesion on mechanical hypersensitivity and escape/avoidance behavior in an animal model of neuropathic pain.
Stacey C. LaGraize,Christopher J. LaBuda,Margaret A. Rutledge,Raymond L. Jackson,Perry N. Fuchs +4 more
TL;DR: Results highlight the utility of novel behavioral test paradigms and provide additional support for the role of the ACC in higher order processing of noxious information, suggesting that ACC lesions selectively decrease negative affect associated with neuropathy-induced hypersensitivity.
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Learned helplessness, inactivity, and associative deficits: Effects of inescapable shock on response choice escape learning
TL;DR: These experiments explored whether exposure to inescapable shock produces a subsequent deficit in the organism's propensity to associate its behavior with shock termination, and found that inescapables shock produced slow learning of the correct choice for escape, even though active choices occurred on every trial.
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Exposure to inescapable shock produces both activity and associative deficits in the rat
TL;DR: It was shown that inescapable shock interfered with the acquisition of signaled punishment suppression but not CER suppression, which is consistent with the possibility that in unavoidable shock may, in addition to reducing activity, produce an associative deficit.
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