5 Papers
Ran Luo is an academic researcher from China Three Gorges University. The author has contributed to research in topics: Cancer research & Medicine. The author has an hindex of 2, co-authored 2 publications.
Chat about Author
Papers
Exosomes from young healthy human plasma promote functional recovery from intracerebral hemorrhage via counteracting ferroptotic injury.
Wenqin Yang,Ning-Fa Ding,Ran Luo,Qian Zhang,Zhenhua Li,Feng-chun Zhao,Shuixian Zhang,Xu-Yang Zhang,Tengyuan Zhou,Haomiao Wang,Long Wang,Shengli Hu,Guixue Wang,Hua Feng,Rong Hu +14 more
TL;DR: Zhang et al. as discussed by the authors showed that exosomes derived from young healthy human plasma exhibiting typical exosome features could facilitate functional recovery of intracerebral hemorrhage (ICH) mice.
60
Tumor-Associated Macrophages: Key Players in Triple-Negative Breast Cancer
TL;DR: Triple negative breast cancer (TNBC) refers to the subtype of breast cancer which is negative for ER, PR, and HER-2 receptors, and tumor-associated macrophages (TAMs) refer to the leukocyte infiltrating tumor, derived from circulating blood mononuclear cells and differentiating into macrophage after exuding tissues.
•Journal Article
Six1 promotes glioblastoma cell proliferation and invasion by upregulation of connective tissue growth factor.
TL;DR: Overexpression of sine oculis homeobox homolog 1 (Six1), a developmental transcription factor implicated in tumor onset and progression, can significantly promote glioblastoma cell proliferation and invasion by upregulating connective tissue growth factor (CTGF).
19
MALT1 regulates Th2 and Th17 differentiation via NF-κB and JNK pathways, as well as correlates with disease activity and treatment outcome in rheumatoid arthritis
Qiubo Wang,Yapeng Wang,Qingyang Liu,Ying Chu,Rui Mi,Feng Jiang,Jingjing Zhao,Ke-yao Hu,Ran Luo,Yufeng Feng,Harrison Lee,Dong Zhou,Jingyi Mi,Ruoyu Deng +13 more
TL;DR: Investigation of the relationship of MALT1 with disease features, treatment outcome, as well as its effect on Th1/2/17 cell differentiation and underlying molecule mechanism in rheumatoid arthritis found it correlates with disease activity and treatment outcome in RA.
TAZ promotes temozolomide resistance by upregulating MCL-1 in human glioma cells.
TL;DR: It is shown that expression of transcriptional co-activator with PDZ-binding motif (TAZ) in glioma cells correlated with temozolomide chemoresistance in human gli cancer cells, and TAZ may provide a promising target for improving the therapeutic outcome of temozoomide-resistant gliomas.