Ralf P. Brandes
Goethe University Frankfurt
306 Papers
2.4K Citations
Ralf P. Brandes is an academic researcher from Goethe University Frankfurt. The author has contributed to research in topics: NADPH oxidase & Biology. The author has an hindex of 77, co-authored 271 publications. Previous affiliations of Ralf P. Brandes include University of California, San Francisco & University of Würzburg.
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Papers
Ncf1 Provides a Reactive Oxygen Species-Independent Negative Feedback Regulation of TLR9-Induced IL-12p70 in Murine Dendritic Cells
Cornelia Richter,Martina Herrero San Juan,Jutta Will,Ralf P. Brandes,Ulrich Kalinke,Shizuo Akira,Josef Pfeilschifter,Malin Hultqvist,Rikard Holmdahl,Heinfried H. Radeke +9 more
TL;DR: Data suggest for the first time an important role for Ncf1 in the fine tuning of the TLR9/MyD88 pathway in vitro and in vivo that is independent of its role as an activator of NOX2.
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Reprogramming of myeloid angiogenic cells by Bartonella henselae leads to microenvironmental regulation of pathological angiogenesis
Fiona O’Rourke,Tanja Mändle,Carmen Urbich,Stefanie Dimmeler,U. Ruth Michaelis,Ralf P. Brandes,Matthias Flötenmeyer,Claudia Döring,Martin-Leo Hansmann,Kirsten Lauber,Wibke Ballhorn,Volkhard A. J. Kempf +11 more
TL;DR: It is demonstrated that manipulation of myeloid cells by pathogenic bacteria can contribute to microenvironmental regulation of pathological tissue growth and suggest parallels underlying both bacterial infections and cancer.
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Dietary L-arginine normalizes endothelin-induced vascular contractions in cholesterol-fed rabbits.
Laddawal Phivthong-ngam,Stefanie M. Bode-Böger,Rainer H. Böger,Michael Böhme,Ralf P. Brandes,Andreas Mügge,Jürgen C. Frölich +6 more
TL;DR: It is suggested that L-arginine restores endothelial function and normalizes the synthesis and vasoconstrictor response to ET-1 in hypercholesterolemia.
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Computational Methods to Study DNA:DNA:RNA Triplex Formation by lncRNAs
TL;DR: A review of the recent progress in the prediction of DNA:DNA:RNA:RNA triplexes can be found in this paper , where different prediction tools (Triplexator, LongTarget, TRIPLEXES, Triplex Domain Finder and TriplexFFP) are discussed.
Increased sensitivity to endothelial nitric oxide (NO) contributes to arterial normotension in mice with vascular smooth muscle-selective deletion of the atrial natriuretic peptide (ANP) receptor.
TL;DR: It is concluded that SMC GC-A KO mice exhibit a higher vasodilating sensitivity to NO, which can be attributed to an enhanced expression of sGC, whereas the expression and/or activity levels of downstream cGMP-effector pathways are not involved.
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