Rajesh Dash
Stanford University
55 Papers
647 Citations
Rajesh Dash is an academic researcher from Stanford University. The author has contributed to research in topics: Medicine & Phospholamban. The author has an hindex of 18, co-authored 48 publications. Previous affiliations of Rajesh Dash include University of California, San Francisco & University of Cincinnati Academic Health Center.
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Papers
Herpes simplex virus vectors overexpressing the glucose transporter gene protect against seizure-induced neuron loss
TL;DR: It is demonstrated that HSV-mediated gene transfer for neuroprotection need not be carried out in anticipation of neurologic crises and an inverse correlation between the extent of vector expression in the dentate and the amount of CA3 damage resulting from the simultaneous delivery of kainic acid is demonstrated.
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Differential regulation of p38 mitogen-activated protein kinase mediates gender-dependent catecholamine-induced hypertrophy
Rajesh Dash,Albrecht Schmidt,Anand Pathak,Michael J Gerst,Danuta Biniakiewicz,Vivek J. Kadambi,Brian D. Hoit,William T. Abraham,Evangelia G. Kranias +8 more
TL;DR: It is suggested that norepinephrine-induced hypertrophy is linked closely with p38 MAP kinase activation, which can be endogenously modulated through estrogen-responsive regulation of MKP-1 expression.
Altering the receptor-effector ratio by transgenic overexpression of type V adenylyl cyclase: enhanced basal catalytic activity and function without increased cardiomyocyte beta-adrenergic signalling.
N M Tepe,John N. Lorenz,Atsuko Yatani,Rajesh Dash,Evangelia G. Kranias,G W Dorn nd,Stephen B. Liggett +6 more
TL;DR: At native stoichiometries, the levels of adenylyl cyclase influence basal activities and cardiac function, but do not constrain betaAR signaling in the cardiomyocyte.
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Astressin, a novel and potent CRF antagonist, is neuroprotective in the hippocampus when administered after a seizure
TL;DR: Astressin fulfills a critical prerequisite for any eventual therapeutic use of CRF antagonists, namely that they need not be administered in anticipation of a neurological insult.
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Herpes simplex virus vector system: analysis of its in vivo and in vitro cytopathic effects
Dora Y. Ho,Sheri L. Fink,Matthew S Lawrence,Timothy J. Meier,Tippi C. Saydam,Rajesh Dash,Robert M. Sapolsky +6 more
TL;DR: It is observed that uninfected cell lysates damaged neurons, both in vivo and in vitro, and purification of the virus from soluble cellular components by a simple pelleting step can significantly decrease such acute toxicity.
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