Qinglin Li
Anhui University
22 Papers
47 Citations
Qinglin Li is an academic researcher from Anhui University. The author has contributed to research in topics: Apoptosis & Biology. The author has an hindex of 13, co-authored 15 publications. Previous affiliations of Qinglin Li include University of Arkansas for Medical Sciences.
Chat about Author
Papers
Involvement of ubiquitin proteasome system in protective mechanisms of Puerarin to MPP+-elicited apoptosis
Yue-Fa Cheng,Guo-Qi Zhu,Mei Wang,Hui Cheng,An Zhou,Ning Wang,Nianbai Fang,Xun-Cui Wang,Xiao-Qiu Xiao,Zhi-Wu Chen,Qinglin Li +10 more
TL;DR: Perarin could protect MPP(+)-induced SH-SY5Y cells from apoptosis by regulating the function of UPS, as evidenced by promoting cell viability, improving morphological changes and reducing apoptotic rate.
94
Gambogenic acid inhibits proliferation of A549 cells through apoptosis-inducing and cell cycle arresting.
Qinglin Li,Hui Cheng,Guo-Qi Zhu,Li Yang,An Zhou,Xiaoshan Wang,Nianbai Fang,Lunzhu Xia,Jingjing Su,Mei Wang,Daiying Peng,Qi Xu +11 more
TL;DR: The results suggested that GNA significantly inhibited the proliferation of several tumor cell lines in vitro and in vivo and might suggest that G NA inhibits tumor cell proliferation via apoptosis-induction and cell cycle arrest.
82
Gambogenic acid induced mitochondrial-dependent apoptosis and referred to phospho-Erk1/2 and phospho-p38 MAPK in human hepatoma HepG2 cells.
Fenggen Yan,Mei Wang,Jiaming Li,Hui Cheng,Jingjing Su,Xiaoshan Wang,Haiyun Wu,Lunzhu Xia,Xiaoxiang Li,Hebron C. Chang,Qinglin Li +10 more
TL;DR: Results indicated that gambogenic acid induced mitochondrial oxidative stress and activated caspases through a caspase-3 and casp enzyme-9-dependent apoptosis pathway and was involved in the phospho-Erk1/2 and phospho -p38 MAPK proteins expression changes in HepG2 cells.
70
Gambogenic acid mediated apoptosis through the mitochondrial oxidative stress and inactivation of Akt signaling pathway in human nasopharyngeal carcinoma CNE-1 cells.
TL;DR: The results indicated that Gambogenic acid mediated apoptosis through inactivation of Akt, accompanied with mitochondrial oxidative stress and cross-talk with Bcl-2 family in the process of apoptosis.
62
Solid lipid nanoparticles as delivery systems for Gambogenic acid.
TL;DR: The freeze-dried powders form could enhance the long-term stability of SLN, and solid lipid nanoparticles encapsulation could effectively strategy to change the poor aqueous solubility and prolong the half-life of GNA.
57