Qing Qi
Chinese Academy of Sciences
9 Papers
6 Citations
Qing Qi is an academic researcher from Chinese Academy of Sciences. The author has contributed to research in topics: Inflammation & Colitis. The author has an hindex of 5, co-authored 8 publications.
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Papers
Artemisinin analogue SM934 ameliorates DSS-induced mouse ulcerative colitis via suppressing neutrophils and macrophages.
Yu-xi Yan,Mei-juan Shao,Qing Qi,Yan-sheng Xu,Xiaoqian Yang,Fenghua Zhu,Shijun He,Pei-Lan He,Chunlan Feng,Yanwei Wu,Heng Li,Wei Tang,Jianping Zuo,Jianping Zuo +13 more
TL;DR: The results reveal the protective effects of SM934 on DSS-induced colitis can be attributed to its suppressing effects on neutrophils and macrophages and its inhibitory role in the NF-κB signaling, suggests thatSM934 might be a potential effective drug for ulcerative colitis.
Inhibition of phosphodiesterase-4 attenuates murine ulcerative colitis through interference with mucosal immunity.
Heng Li,Chen Fan,Chunlan Feng,Yanwei Wu,Huimin Lu,Pei-Lan He,Xiaoqian Yang,Fenghua Zhu,Qing Qi,Yuanzhuo Gao,Jianping Zuo,Wei Tang +11 more
TL;DR: This study was designed to explore whether PDE4 inhibition by apremilast exerts protective effects in dextran sulfate sodium‐induced murine UC.
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RIPK1 inhibitor ameliorates colitis by directly maintaining intestinal barrier homeostasis and regulating following IECs-immuno crosstalk.
Huimin Lu,Heng Li,Chen Fan,Qing Qi,Yu-xi Yan,Yanwei Wu,Chunlan Feng,Bing Wu,Yuanzhuo Gao,Jianping Zuo,Wei Tang +10 more
TL;DR: It is demonstrated that RIPK1 inhibitor could ameliorate the intestinal barrier injury by reducing tight junctions' disruption and accompanying oxidative stress and exerts suppressive function in intestinal inflammatory response.
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Identification of phosphodiesterase-4 as the therapeutic target of arctigenin in alleviating psoriatic skin inflammation
Heng Li,Xianglei Zhang,Caigui Xiang,Chunlan Feng,Chen Fan,Moting Liu,Huimin Lu,Haixia Su,Yu Zhou,Qing Qi,Yechun Xu,Wei Tang +11 more
TL;DR: Proteomics analysis revealed that arctigenin could rectify the immune dysfunction and hyperactivation of keratinocytes in the inflamed skin microenvironments, which might be largely related to the expression of Keratins.
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Triptolide analog LLDT-8 ameliorates psoriasis-like dermatitis in BALB/c mice via suppressing the IL-36α signaling pathway.
Qing Qi,Qian Li,Qian Li,Hongwen Zhu,Huimin Lu,Xiaoqian Yang,Yanwei Wu,Chunlan Feng,Chen Fan,Heng Li,Bing Wu,Yuanzhuo Gao,Zongwang Zhang,Zongwang Zhang,Hu Zhou,Jianping Zuo,Wei Tang +16 more
TL;DR: Li et al. as mentioned in this paper investigated the effects and underlying mechanisms of (5R)-5-hydroxytriptolide (LLDT-8) on a murine psoriasis-like dermatitis model and related cell lines.
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