Qing Li
East China Normal University
5 Papers
5 Citations
Qing Li is an academic researcher from East China Normal University. The author has contributed to research in topics: Experimental autoimmune encephalomyelitis & Innate immune system. The author has an hindex of 2, co-authored 4 publications.
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Papers
Bcl6 modulates innate immunity by controlling macrophage activity and plays critical role in experimental autoimmune encephalomyelitis
Qing Li,Lei Zhou,Ling Wang,Shiqiang Li,Guiliang Xu,Haijuan Gu,Dali Li,Mingyao Liu,Lei Fang,Zhengyi Wang,Shuhua Han,Biao Zheng,Biao Zheng +12 more
TL;DR: It is demonstrated that EAE is exacerbated in LysM Cre+/− Bcl6fl/fl mice and it is found that the disease pathology is mainly associated with a biased M1 macrophage activity and an enhanced encephalitogenic CD4+ Th17 cell response.
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Lipid Receptor G2A-Mediated Signal Pathway Plays a Critical Role in Inflammatory Response by Promoting Classical Macrophage Activation.
Qing Li,Chunlei Feng,Lingyun Li,Guiliang Xu,Haijuan Gu,Shiqiang Li,Dali Li,Mingyao Liu,Shuhua Han,Biao Zheng +9 more
TL;DR: In this article, the effects of lipoprotein-associated phospholipase A2 (Lp-PLA2) on M1 function were mediated by lysophosphatidylcholine, a bioactive product of oxidized lipids hydrolyzed by PLA2 through JAK2-independent activation of STAT5 and upregulation of IRF5.
6
The combination of gemcitabine and ginsenoside Rh2 enhances the immune function of dendritic cells against pancreatic cancer via the CARD9-BCL10-MALT1 / NF-κB pathway.
Qing Li,Jialuo He,Senlin Li,Cheng Tian,Jian Yang,Hui-Min Yuan,Yi Lu,Paolo Fagone,Ferdinando Nicoletti,Ming Xiang +9 more
TL;DR: In this article , the authors showed that after GEM-Rh2 therapy, there was significantly greater tumor infiltration by dendritic cells (DCs), and that the activation of DCs may remodel the cold tumor immune microenvironment (TIME) and optimize GEM chemotherapy for future therapeutic use.
REGγ controls Th17 cell differentiation and autoimmune inflammation by regulating dendritic cells.
Lei Zhou,Liangfang Yao,Qing Zhang,Wei Xie,Xiaoshuang Wang,Huihui Zhang,Jinjin Xu,Qingxia Lin,Qing Li,Yang Xuan,Lei Ji,Lu Wang,Weicang Wang,Weichao Wang,Tingting Shi,Lei Fang,Biao Zheng,Lei Li,Shuang Liu,Bianhong Zhang,Xiaotao Li,Xiaotao Li +21 more
TL;DR: A previously unknown mechanism by which REGγ-mediated protein degradation in DCs controls the differentiation of Th17 cells and the onset of an experimental autoimmune disease is delineated.
Docosahexaenoic acid ameliorates autoimmune inflammation by activating GPR120 signaling pathway in dendritic cells.
TL;DR: In this paper, the effect of omega-3 polyunsaturated fatty acids (n-3 PUFAs) on the functional activities of dendritic cells (DCs) was investigated in a model for multiple sclerosis (MS) and other autoimmune diseases.