Qin Wei
8 Papers
Qin Wei is an academic researcher. The author has contributed to research in topics: Messenger RNA & Cancer research. The author has an hindex of 1, co-authored 1 publications.
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Papers
METTL3 preferentially enhances non-m6A translation of epigenetic factors and promotes tumourigenesis
Xueju Wei,Yue Huo,Jingnan Pi,Yufeng Gao,Shuan Rao,Manman He,Qin Wei,Peng Song,Yiyin Chen,Dongxu Lu,Wei Song,Ju-Fang Liang,Lin Gui Xu,Hai-Xu Wang,Guolin Hong,Yuehong Guo,Yanmin Si,Jiayue Xu,Xiaoshuang Wang,Yanni Ma,Shuyang Yu,Dongling Zou,Jing Jin,Fang Wang,Jia Yu +24 more
TL;DR: The cytoplasmic METTL3 enhances the translation of epigenetic mRNAs, thus serving as an oncogenic driver in cancer progression, andMETTL3 subcellular distribution can assist diagnosis and predict prognosis for patients with cancer.
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Upregulation of LRRC8A by m5C modification-mediated mRNA stability suppresses apoptosis and facilitates tumorigenesis in cervical cancer
Yanjie Chen,Xinzhao Zuo,Qin Wei,Jie Xu,Xiaoyi Liu,Shiling Liu,Haocheng Wang,Qingya Luo,Yuya Wang,Y. Yang,Hongyan Zhao,Jing Xu,Tao Liu,Ping Yi +13 more
TL;DR: In this article , the authors found that LRRC8A is upregulated by NSUN2-mediated m5C modification, which is associated with poor prognosis in cervical cancer.
NAT10-mediated RNA acetylation enhances HNRNPUL1 mRNA stability to contribute cervical cancer progression
TL;DR: In this paper , the role of NAT10-mediated RNA acetylation modification in cervical cancer remains to be elucidated, and the authors demonstrate that NAT10 contributes to cervical cancer progression by enhancing HNRNPUL1 mRNA stability via ac4C modification.
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RNA m5C modification upregulates E2F1 expression in a manner dependent on YBX1 phase separation and promotes tumor progression in ovarian cancer.
Xiaoyi Liu,Qin Wei,Chenyue Yang,Hongyan Zhao,Jie Xu,Youchaou Mobet,Qingya Luo,Dan Yang,Xinzhao Zuo,Ningxuan Chen,Yu Yang,Li Li,Wei Wang,Jianhua Yu,Jing Xu,Tao Liu,Ping Yi +16 more
TL;DR: RNA m5C modification, facilitated by NSUN2, upregulates E2F1 expression through a positive feedback loop with YBX1 phase separation, promoting ovarian cancer progression by regulating oncogenes MYBL2 and RAD54L.
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AKAP8 promotes ovarian cancer progression and antagonizes PARP inhibitor sensitivity through regulating hnRNPUL1 transcription
Youchaou Mobet,Haocheng Wang,Qin Wei,Xiaoyi Liu,Dan Yang,Hongyan Zhao,Yu Yang,Rosalie Anne Ngono Ngane,Jacob Souopgui,Jing Xu,Tao Liu,Ping Yi +11 more
TL;DR: AKAP8 promotes ovarian cancer progression by regulating hnRNPUL1 transcription, antagonizing PARP inhibitor sensitivity, and modulating PARP1 expression, suggesting AKAP8 as a potential therapeutic target for improving ovarian cancer treatment.
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