17 Papers
33 Citations
Qin Hao is an academic researcher from University of Texas Health Science Center at Tyler. The author has contributed to research in topics: Kinase & Phosphorylation. The author has an hindex of 10, co-authored 17 publications.
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Papers
Inactivation of SRC family tyrosine kinases by reactive oxygen species in vivo.
TL;DR: H2O2 at low to moderate concentrations markedly inactivated Src family tyrosine kinases temporally and spatially in vivo but not in vitro and rendered endothelial cells resistant to growth factors and cytokines and protected the cells from inflammatory activation.
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•Journal Article
Protein kinases D2 and D3 are novel growth regulators in HCC1806 triple-negative breast cancer cells.
TL;DR: Targeting PKD instead of all PKCs effectively inhibited cell proliferation in a number of breast cancer cell lines and identified PKD2 and PKD3, especially PKD 3, as novel cell growth regulators in HCC1806 triple-negative breast cancer cells.
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Tyrosine phosphatase PTP-MEG2 negatively regulates vascular endothelial growth factor receptor signaling and function in endothelial cells.
TL;DR: It is found that PTP-MEG2 is a negative regulator of VEGFR2 signaling and function in ECs, and its function in endothelial cells (ECs) is investigated.
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Transcription Factor Runx3 Is Induced by Influenza A Virus and Double-Strand RNA and Mediates Airway Epithelial Cell Apoptosis
TL;DR: This study found for the first time that transcription factor Runx3, a developmental regulator and tumor suppressor, was induced by IAV H1N1 and H3N2, viral RNA, a synthetic analog of viral double-stranded RNA (dsRNA) polyinosinic-polycytidylic acid, and type-II interferon-γ (IFNγ) in human airway epithelial cells.
Enhanced RIPK3 kinase activity-dependent lytic cell death in M1 but not M2 macrophages.
TL;DR: It is demonstrated that M1 but not M2 subtypes of macrophages are more susceptible to inflammation-related lytic cell death in an RIPK3 kinase activity-dependent manner.
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