Per Svenningsson
Karolinska Institutet
447 Papers
2.3K Citations
Per Svenningsson is an academic researcher from Karolinska Institutet. The author has contributed to research in topics: Medicine & Parkinson's disease. The author has an hindex of 72, co-authored 393 publications. Previous affiliations of Per Svenningsson include Karolinska University Hospital & Rockefeller University.
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Papers
Genetic evidence for role of integration of fast and slow neurotransmission in schizophrenia.
Anna Devor,Ole A. Andreassen,Ole A. Andreassen,Yunpeng Wang,Yunpeng Wang,Yunpeng Wang,Tuomo Mäki-Marttunen,Tuomo Mäki-Marttunen,Olav B. Smeland,Olav B. Smeland,Chun Chieh Fan,Andrew J. Schork,Dominic Holland,Wesley K. Thompson,Aree Witoelar,Aree Witoelar,Chi-Hua Chen,Rahul S. Desikan,Linda K. McEvoy,Srdjan Djurovic,Paul Greengard,Per Svenningsson,Gaute T. Einevoll,Gaute T. Einevoll,Anders M. Dale +24 more
TL;DR: These findings extend the existing pathophysiology hypothesis by suggesting a unifying role of dysregulation in neuronal excitability and synaptic integration in SCZ, and support the concept of SCZ as an ‘associative’ disorder.
Neurogenic effects of fluoxetine are attenuated in p11 (S100A10) knockout mice
TL;DR: In response to fluoxetine, p11 KO mice have reduced cell proliferation, neurogenesis, cell survival, and cell apoptosis in the subgranular zone of hippocampus when compared with WT littermates.
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Opposite tonic modulation of dopamine and adenosine on c-fos gene expression in striatopallidal neurons.
TL;DR: It is shown that an acute decrease of dopamine release causes an induction of c-fos messenger RNA in dopamine D2 receptor-containing striatopallidal neurons, and that endogenous dopamine and adenosine exert opposite effects on the activity of the D2-containingstriatop allidal neurons.
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Mass Spectrometry Imaging, an Emerging Technology in Neuropsychopharmacology
TL;DR: The results presented demonstrate that mass spectrometry imaging is a useful experimental method with diverse applications in neuroscience and its potential for neuroscience research is discussed.
92
Antidepressants reverse the attenuation of the neurotrophic MEK/MAPK cascade in frontal cortex by elevated platform stress; reversal of effects on LTP is associated with GluA1 phosphorylation.
Hongshi Qi,François Mailliet,François Mailliet,Michael Spedding,Cyril Rocher,Cyril Rocher,Xiaoqun Zhang,Philippe Delagrange,Bruce S. McEwen,Thérèse M. Jay,Thérèse M. Jay,Per Svenningsson +11 more
TL;DR: Results indicate that acute elevated platform stress down-regulates a putative BDNF/MEK/MAPK signaling cascade in the frontal cortex in a manner that is reversible by the antidepressants tianeptine and imipramine.
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