Paul Greengard
Rockefeller University
909 Papers
20.6K Citations
Paul Greengard is an academic researcher from Rockefeller University. The author has contributed to research in topics: Protein kinase A & Phosphorylation. The author has an hindex of 104, co-authored 900 publications. Previous affiliations of Paul Greengard include Johns Hopkins University School of Medicine & Karolinska Institutet.
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Papers
Erratum: Nitric oxide regulates synaptic transmission between spiny projection neurons (Proc Natl Acad Sci USA (2014) 111 49 (17636-17641) DOI: 10.1073/pnas.1420162111)
Yotam Sagi,Myriam Heiman,Jayms D. Peterson,Sergei Musatov,Mariangela Scarduzio,Stephen M. Logan,Michael G. Kaplitt,Dalton James Surmeier,Nathaniel Heintz,Paul Greengard +9 more
1
Patent
Methods of treating dopamine dysregulation using agents that regulate phosphorylation/dephosphorylation in dopamine signaling
James A. Bibb,Paul Greengard +1 more
- 13 Oct 2000
TL;DR: In this paper, it was revealed that DARPP-32 is a substrate for the cyclin dependent kinase Cdk5 and that the phosphorylation takes place at a specific threonine residue of DARPP32 (Threonine 75).
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Encoding of an engram for food location by satiety-promoting Drd2 hippocampal neurons
Estefania P. Azevedo,Lisa E. Pomeranz,Jia Cheng,Marc Schneeberger,Sarah A. Stern,Katherine R. Doerig,Paul Greengard,Jeffrey M. Friedman +7 more
TL;DR: It is reported that hippocampal dopamine 2 receptor (D2R) neurons are specifically activated by food and that modulation of their activity reduce food intake in mice and identifies a LEC->Hippocampus->Septal high-order circuit that encodes the memory of food location.
Neuron-Glia Signaling Regulates the Onset of the Antidepressant Response
Vicky Yao,Ammar Aly,Salina Kalik,Jodi Gresack,Wei Wang,Annie Handler,Anne Schaefer,Olga G. Troyanskaya,Paul Greengard,Revathy U. Chottekalapanda +9 more
TL;DR: In this paper, a single cell sequencing approach was used to analyze gene expression changes in mice subjected to stress-induced depression and determined their temporal response to antidepressant treatment in the cerebral cortex.