Paul B. Rainey
Max Planck Society
235 Papers
1.2K Citations
Paul B. Rainey is an academic researcher from Max Planck Society. The author has contributed to research in topics: Biology & Pseudomonas fluorescens. The author has an hindex of 70, co-authored 222 publications. Previous affiliations of Paul B. Rainey include Massey University & Mansfield University of Pennsylvania.
Chat about Author
Papers
Eco-evolutionary feedback and the tuning of proto-developmental life cycles.
Eric Libby,Paul B. Rainey +1 more
TL;DR: A model in which environmental fluctuations are coupled to population growth generates stable selection for a single optimal strategy that is largely insensitive to environmental conditions, including the number of competitors, carrying capacity of the environment, difference in growth rates among phenotypic variants, and population density.
Ribosome Provisioning Activates a Bistable Switch Coupled to Fast Exit from Stationary Phase.
Philippe Remigi,Philippe Remigi,Gayle C. Ferguson,Ellen McConnell,Silvia De Monte,Silvia De Monte,David W. Rogers,Paul B. Rainey,Paul B. Rainey,Paul B. Rainey +9 more
TL;DR: It is shown that pyrimidine limitation triggers an increase in ribosome biosynthesis and that switching is caused by competition between ribosomes and CsrA/RsmA proteins for the mRNA transcript of a positively autoregulated activator of colanic acid biosynthesis.
26
The genetic structure of Staphylococcus aureus populations from the Southwest Pacific.
TL;DR: With the exception of CC75 strains, the global collection of S. aureus encompasses relatively little diversity, with novel STs arising locally from a small number of widespread lineages.
Curiosities of REPINs and RAYTs
Frederic Bertels,Paul B. Rainey +1 more
TL;DR: Factors affecting the evolution of REPIN diversity, the ease with which REPINs might be co-opted by host genomes and the consequences of RE pin activity for the structure of bacterial genomes are considered.
Environmentally constrained mutation and adaptive evolution in Salmonella
TL;DR: It is shown that the mutation is not 'directed' and described its genetic basis, and compelling evidence is presented in support of the hypothesis that the mutational event is constrained by signals transmitted from the external environment via changes in the activity of DNA gyrase.
22