Paul A. Smith

TL;DR: It was shown, in the clinical setting, that central sympathetic hyperactivity exists in white-coat hypertension, albeit to a lesser degree than EHT, suggesting that WHT may not be entirely benign and that the observed sympatheticHyperactivity may be responsible for development of target organ damage in this group of patients.

TL;DR: In conclusion, both UA and AMI result in sympathetic hyper-activity, although this is of smaller magnitude in UA and is less protracted than in AMI.

TL;DR: Findings suggest that the worse prognosis after ant-AMI compared with after inf-AMI would not be related primarily to the degree of sympathetic hyperactivity.

TL;DR: Investigating the relationship between neurovascular compression of the rostral ventrolateral medulla (RVLM) and the magnitude of central sympathetic hyperactivity in normal subjects and in patients with untreated and uncomplicated essential hypertension found increased sympathetic activity was strongly predictive of NVC.